Re: Minimalist Yeast Abatement Protocol

One of the noticeable effects of starting to tackle the yeast problem in the body is the rapidity with which symptoms appear, disappear, and reappear amongst different locales. The polymorphism of Candida Albicans certainly allows it access to all parts of the body and we have already looked at the idea of polysystemic colonization even in otherwise healthy individuals:

See "Eye Floaters" //www.curezone.org/forums/fm.asp?i=1961901

The rapid "shifting symptom" syndrome suggests that this dissemination has already occurred prior to the start of an anti-yeast protocol. While it might seem that you are physically chasing it around the body, rather when you challenge the yeast in one location, colonies in other locations may become more active to take up the slack. Attack it in the gut with antifungals, and blood-brain barrier symptoms flare up. From a yeast point of view, this is a survival technique. If it is too aggressive in its usage of acetaldehyde for host control, then it may kill off its visible means of support and cease to exist. If its own population drops too low, then its survival is threatened -- a tenuous balancing act.

We have already seen that Candida Albicans is responsive to energetic signalling techniques using homeopathic sulfur that can alter its morphic state (budding versus hyphal) and postulated that some form of energetic quorum-sensing technique is being utilized by this yeast to control its own virulence through regulation of its acetaldehyde production amongst different colonization sites in the body:

See "Homeopathic Sulfur + Quorum-Sensing" //www.curezone.org/forums/fm.asp?i=1962569

This is no dumb microbe that falls over dead when you clobber it on the head. In some sense, we are not "bodies with yeast" but "yeast with bodies".

Given its omnipresence, it would not be surprising to find other striking examples of this quorum-sensing activity as the human mind struggles with maintaining the health of its own cells that are constantly under duress from an organism using acetaldehyde emission as a biochemical intimidator.

Some studies have gone so far as to suggest that "acetaldehydism" is a better descriptive term than "alcoholism" [1] indicating that the driving force underlying the addiction is the desire for the acetaldehyde byproduct of alcohol rather than the ethanol itself. This research has focused on the neurotransmitter-mediated addictive effects of acetaldehyde. However, anecdotal reports on the web have indicated the loss of alcohol cravings after the consumption of substances (e.g. fulvic acid) that would appear to be functioning as an acetaldehyde scavenger. If the mind/body interface is actually addicted to acetaldehyde, why would an acetaldehyde scavenger reduce the cravings for the precursor of acetaldehyde production? Perhaps there is more happening here…

When acetaldehyde appears in the liver as a byproduct of alcohol metabolism, the body is normally equipped with ample supplies of hepatic aldehyde dehydrogenase enzymes that quickly convert it to acetic acid. When acetaldehyde is emitted from yeast colonization sites, it is much more likely to be a precipitator of biochemical tissue damage, and ultimately, disease before the body is able to neutralize it. Furthermore, if yeast colonies down-regulate their own emission of acetaldehyde when the level of acetaldehyde in some other part of the body increases, then the appearance of acetaldehyde in the liver may be sensed by yeast colonization as an energetic message indicating the increase of activity of "another yeast colony". The effect would be that the yeast in the body temporarily ceases its own production of acetaldehyde until the concentration drops once again.

So now we have a model where the mind has found that it can minimize the irritating release of toxic acetaldehyde in places of the body where it cannot adequately deal with it by increasing the concentration of acetaldehyde in the liver where it can process it. And the technique that the mind uses to achieve this is through cravings -- alcohol cravings. Whenever the hepatic concentration drops and the yeast starts up its fermentative release mechanisms once again, the craving mechanism for the relief valve induces more alcohol consumption.

Since the alcohol intake does not actually remove the yeast from the picture, the cycle of using alcohol consumption for symptom remission becomes chronic. This overloads the hepatic mechanisms for dealing with ingested alcohol/acetaldehyde, with debilitating alcohol-generated intoxication and acetaldehyde-generated hangover symptoms, the unfortunate result. When an oral acetaldehyde scavenger is utilized, this mops up the yeast-released acetaldehyde and the alcohol to acetaldehyde pathway is no longer required, so the cravings for alcohol abate.

Disulfiram and calcium carbimide are used in the treatment of alcoholism as aldehyde dehydrogenase inhibitors which can block the detoxification of acetaldehyde generated in the processing of alcohol in the liver. They create such high bloodstream levels of acetaldehyde that the accompanying sweating, breathing difficulty, rapid heartbeat, rash, nausea/vomiting, and headache symptoms create an aversion to alcohol intake. These substances are an attempt, essentially, to undermine the association that the brain has made between alcohol consumption and relief from yeast aggravation. However, they do nothing to address the original source of the acetaldehyde in this model (on-board commensal yeast) and put the remainder of the body at risk to exposure to additional amounts of this toxic substance from any source.

Does this mean that a yeast abatement protocol that both scavenges and reduces the production of yeast-released acetaldehyde might be beneficial in such a situation? Perhaps, but there is a simple technique for testing this hypothesis in individuals who are prone to these cravings. Whenever the desire for alcohol appears, invoke an energetic homeopathic acetaldehyde (30C) visualization [2] and see if the craving abates. If it does, you have tricked the yeast into thinking that there is additional acetaldehyde production in the body without actually ingesting or creating any of the actual toxic substance! Then you also know the real source of the problem...


[1] Cannizzaro C et al., "Role of acetaldehyde in alcohol addiction: current evidence and future perspectives", Malta Medical Journal, 2011: 3; 27.
http://www.um.edu.mt/umms/mmj/abstract.php?article=330

[2] "Homeopathy Without The Pellets" in "Astrophysiology… and Yeast", 2011.
http://www.scribd.com/doc/74090699
http://www.epubbud.com/book.php?g=7JQU45V8

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