Re: Minimalist Yeast Abatement Protocol

Given the analysis of the potential of acetaldehyde autoxidation byproducts:

See "Acetaldehyde + Pulmonary Peroxidosis" //www.curezone.org/forums/fm.asp?i=1984937

it should go without saying that using something as toxic as hydrogen peroxide in a treatment protocol is a bit like throwing starter fluid onto a smouldering barbecue. There may be some temporary remission or change of symptoms because of the antimicrobial and acetaldehyde neutralizing properties, but this is not something that can be continued in the long term without sustaining collateral damage resulting from both the hydrogen peroxide itself and the side reactions that it can precipitate.

Hydrogen peroxide (H2O2) will react with acetaldehyde (CH3CHO) in aqueous (H2O) solution producing hydroxy alkyl peroxides [1] such as 1-hydroxyethyl hydroperoxide, CH3CH(OH)OOH, which is unstable and may produce damaging free radical species:

CH3CHO + H2O2 (Hydrogen-Peroxid) <--> CH3CH(OH)OOH

As mentioned in the referenced post, hydrogen peroxide can also react with acetic acid (CH3COOH) to produce peracetic acid (CH3COOOH), an oxidant with similar potency to chlorine dioxide:

CH3COOH + H2O2 (Hydrogen-Peroxid) <--> CH3COOOH + H2O

Acetic acid is the body's byproduct of acetaldehyde dehydrogenation. Microbes in the body's bioflora also produce acetic acid [2] that would be bioavailable for the hydrogen peroxide reaction to peracetic acid.

We have seen chlorine dioxide before as a toxic byproduct in the reaction of sodium chlorite (MMS) with acetaldehyde:

See "Acetaldehyde + Miracle-Mineral-Supplement " //www.curezone.org/forums/fm.asp?i=1910030

Peroxides in general are unstable since the oxygen-oxygen chemical bond can easily cleave forming reactive radical species. Not only are they irritating and inflammatory, they are toxic to living cells resulting from the oxidation of proteins, membrane lipids and DNA. Using hydrogen peroxide therapy can easily set the pulmonary peroxidosis cascade in motion as well as bleaching essential organs (such as the liver) if the mixture concentration isn't just right [3].

Since yeast-released acetaldehyde is implicated in this series in so many different disease processes, then the strategy of using a toxic substance (sodium chlorite or hydrogen peroxide) to kill the yeast or reduce acetaldehyde exposure by diverting it into another substance will produce results but at what cost? Now that we know more about the why's and wherefore's of the disease process in relation to yeast and acetaldehyde, there are less risk-prone approaches for addressing the difficulty. It's not an easy task, by any means, but there is a way out of the maze using techniques that can be continued indefinitely [4].

[1] Satterfield et al., "Reaction of Aldehyde and Hydrogen Peroxide in Aqueous Solution",Ind. Eng. Chem., 1954, 46 (5), pp 998–1001
http://pubs.acs.org/doi/abs/10.1021/ie50533a049

[2] Henningsson A, "Short-chain fatty acid formation at fermentation of indigestible carbohydrates.", Food & Nutrition Research 2001 Vol 45
http://www.foodandnutritionresearch.net/index.php/fnr/article/view/1801

[3] Pritchett S et al., "Accidental ingestion of 35% hydrogen peroxide", Can J Gastroenterol. 2007 October; 21(10): 665–667.
http://www.ncbi.nlm.nih.gov/pmc/articles/PMC2658134

[4] "Yeast Begone!" in "Astrophysiology... and Yeast", 2011.
http://www.scribd.com/doc/74090699
http://www.epubbud.com/book.php?g=7JQU45V8