Re: Update! Eyesight improving, and general improvements with some side effects from Iodine and Borax protocols

Your emotionally charged response was unwarranted and indicates your inherent bias.

Someone else took the forum off topic by suggesting supplementation of retinol. I study retinol as part of my academic work and responded with a word of caution.

Retinol's counterparts are the other fat-soluble vitamins, particularly vitamin D, but also vitamins E and K1/K2. There is an intricate relationship between all 4.

Your assertion that I asserted that retinol can't act as a terminator is a strawman argument. I never said this. Re-read my post.

Retinol is very much like the polyunsaturated fats in the manner in which I already characterized it. Both are extremely unstable because of their richness in double bonds. This is precisely why retinol binding protein exists. It encases the retinol molecule to prevent its premature and inappropriate oxidation. Free retinol is toxic and this has been demonstrated thoroughly in the literature.

The notion that polyunsaturated fats (PUFA) are necessary for life needs clarification. PUFAs are necessary for some organisms but not so for others, or if they are they are required in such minuscule amounts as to render a deficiency unlikely with a normal diet. The studies upon which this idea is based are flawed. The Burr studies that are often cited as the first evidence of "essential fatty acids" were debunked later after the discovery of vitamin B6. The rat acrodynia that developed in response to a polyunsaturated fat "deficiency" was later cured by adding vitamin B6 to fat-free chow. The mechanism by which polyunsaturated fat reverses the acrodynia is by sparing vitamin B6. Thus, the true deficiency is in vitamin B6, not PUFAs.

I believe you need to study evolution a little more closely. In a retinol-poor environment, the carotene-conversion pathway would have been selected for because it would have given those people a reproductive advantage over those who could not convert carotene. Additionally, those who could absorb and store retinol more efficiently would have been selected for because this also would have given them a reproductive advantage. The carotene conversion pathway exists because at some point in our evolution there was a mutation in an individual allowing them to convert carotene into retinol. This was selected for over time and those who could not do it were selected against because retinol was so rare in the environment. This is how natural selection works. I am a biochemist. I have spent years studying this. Taking people who evidentially evolved in a retinol-poor environment and proceeding to fortify foods and recommend foods naturally high in retinol along with vitamin supplements will cause both acute and chronic toxicities