Re: WARNING - Faulty information being passed around

While I am not an expert, there are large stongyloides and Small Strongyles in horses: The strongyloides can be 30 inches in horses, and the Strongyles are much smaller. I believe it is the smaller version that is a problem, mainly due to the filarial component.

Strongyles migrate mainly in the wall of the intestine. This causes a weakening and thinning of the horse intestine wall.

In humans:

A number of nematodes infect the intestine.

http://www.ncbi.nlm.nih.gov/books/NBK8261/


A large accumulation of Ascarids may cause obstruction and rupture, Ascaris can cause rupture of the apendicitis, Taenia saginata can cause peritonitis, or sepsis. Strongyloides lead to a thinning of the human intestine epithelial wall...

Strongyloides in humans:

Dissemination occurs upon larval migration to organs beyond the range of the pulmonary phase, such as the liver, heart, lymph nodes, gallbladder, kidneys, pancreas, and brain (Keiser & Nutman, 2004).

Petechiae and purpura have also been reported in disseminated cases as a result of larval migration through vessel walls, which promotes haemorrhage (Basile et al., 2010). Others complications of disseminated strongyloidiasis can occur and include syndromes such as cholecystitis, pancreatitis, paralytic ileus, intestinal perforation or infarction, peritonitis, and sepsis (Kris hnan et al., 2006)

In recent decades, hyperinfection syndrome has increased significantly with the use of immunosuppressive drug therapy.

Corticosteroid s are widely prescribed drugs with potent immunosuppressive effects and are a major risk factor for the transformation of chronic strongyloidiasis into hyperinfection, which has a higher index of mortality (Armignacco et al., 1989; Al Maslamani et al., 2009).







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