Re: Calling All Experts

Incredible.

Sounds like he (and she) is in good, no - great hands. That's some progressive ND/protocol.

Below is a compilation I pulled together on the supplements. It's pretty basic. The Rife reccommendation - fantastic. All the way. Vitamin C IV? I did one but opted for some others. Another good IV which might be even more beneficial could be Glutiathione. Not sure if the two are counterindicative, but many of these type of IV's can be done within a half hour of one another, which is nice.

I'm sure Newport will offer up some good lit related to Lyme and using a function/frequency generator such as a Rife machine. Gosh - wonder who this ND expects will have the ability to Rife him. Must have someone in mind - and this is just wonderful in itself.

And get him on some iodine! What a great post - sounds like a best case scenario in terms of treatment and practioners.

Here's that list on the rest.

Here's some googled info on the sups:

Phosphatidyl lecithin


http://www.evitamins.com/healthnotes.asp?ContentID=2873001


Lecithin/Phosphatidyl Choline
Also Known As: Choline
Introduction
What it does
Where found
Helpful for
Are you deficient?
Amount to take
Side Effects & Interactions
References
What is it?
When medical researchers use the term “lecithin,” they are referring to a purified substance called phosphatidyl choline (PC) that belongs to a special category of fat-soluble substances called phospholipids.

Phospholipids are essential components of cell membranes. Supplements labeled as “lecithin” usually contain 10–20% PC. Relatively pure PC supplements are generally labeled as “phosphatidylcholine.” PC best duplicates supplements used in medical research.

Choline by itself (without the “phosphatidyl” group) is also available in foods and supplements. In high amounts, however, pure choline can make people smell like fish, so it’s rarely used, except in the small amounts found in multivitamin supplements.

What does lecithin/phosphatidyl choline do?
PC acts as a supplier of choline, which is needed for cell membrane integrity and to facilitate the movement of fats in and out of cells. It is also a component of the neurotransmitter acetylcholine and is needed for normal brain functioning, particularly in infants. Although the human body can synthesize choline, additional amounts from the diet are considered essential under certain circumstances. For this reason, PC has been used in a number of preliminary studies for a wide variety of neurological and psychiatric disorders, though not every study suggests that supplemental choline is capable of reaching the brain.1 Choline participates in many functions involving cellular components called phospholipids.

Where is lecithin/phosphatidyl choline found?
Choline, the major constituent of PC, is found in soybeans, liver, oatmeal, cabbage, and cauliflower. Soybeans, egg yolks, meat, and some vegetables contain PC. Lecithin (containing 10–20% PC) is added to many processed foods in small amounts for the purpose of maintaining texture consistency.

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KandidaPlex


http://www.vrp.com/productpage.aspx?ProdID=1670


This powerful phytonutrient complex was designed for intestinal and systemic yeast imbalance.* Along with undecylenic acid, this formula also provides berberine, citrus seed extract and other synergistic factors.
Recommended Dosage: one to two capsules per day, increase by one capsule every two to three days until desired results are achieved (most commonly six per day).

Supplement Facts
Serving Size:1 capsule

Amount Per Serving % Daily Value

Biotin 150 mcg 50

sorbic acid 25 mg *

Undecylenic acid 100 mg *

Pau darco 150 mg *
(Tabebuia heptaphylla) extract (bark)

Grapefruit Seed Extract 40 mg *
(Citrus grandis) (seed) (50% polyphenols)

Berberine (Berberine sulfate) 40 mg *

*Daily Value not established
Other Ingredients:
Microcrystalline cellulose and Hydroxypropyl methylcellulose (Vcap).

Contains no added starch, salt, wheat, gluten,
corn, coloring, or dairy products.
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Conjugated Linoleic Acid

Health Benefits of Conjugated Linoleic Acid


http://www.mercola.com/beef/cla.htm


Many people are taking CLA as an expensive supplement for the benefits described below. But wouldn't it make much more sense to get it for free in your food?

There is a new reason why it may be beneficial to allow cows to graze on pasture. That reason involves a compound called conjugated linoleic acid (CLA).

CLA is a fatty acid found in beef and dairy fats. Scientific interest in CLA was stimulated in 1988 when a University of Wisconsin researcher discovered its antioxidant properties in a study of rats fed fried hamburger. CLA cannot be produced by the human body, but it can be obtained through foods such as whole milk, butter, beef, and lamb.

"The interesting thing is that dairy cattle that graze produce higher amounts of CLA in their milk than those which receive conserved feed, such as grain, hay, and silage," says Agricultural Research dairy scientist Larry Satter. This is true even when the nongrazers eat pasture grass conserved as hay.

Satter, who is based at the Dairy Forage Research Center in Madison, Wisconsin, conducted a study comparing the amount of CLA in milk from cows grazing on pasture to the amount from cows fed hay or silage.

His findings:

Pasture-grazed cows had 500% more CLA in their milk than those fed silage.

Larry Satter is at the USDA-ARS U.S. Dairy Forage Research Center, 1950 Linden Lane, University of Wisconsin, Madison, WI 53706; phone (608) 264-5353, fax (608) 264-5147.

CLA may be one of the most potent antioxidant substances in our diet.

In animal studies, as little as one half of one percent CLA in the diet showed promising health benefits.

In a preliminary study, CLA has also been shown to reduce body fat in people who are overweight.

by Mary Shomon

A double-blind, randomized, placebo-controlled study, published in the December 2000 issue of the Journal of Nutrition found that CLA reduces fat and preserves muscle tissue. According to the research project manager, an average reduction of six pounds of body fat was found in the group that took CLA, compared to a placebo group.

The study found that approximately 3.4 grams of CLA per day is the level needed to obtain the beneficial effects of CLA on body fat.

Dr. Michael Pariza, who conducted research on CLA with the University of Wisconsin-Madison, reported in August 2000 to the American Chemical Society that "It doesn't make a big fat cell get little. What it rather does is keep a little fat cell from getting big."

Pariza's research did not find weight loss in his group of 71 overweight people, but what he did find was that when the dieters stopped dieting, and gained back weight, those taking CLA "were more likely to gain muscle and not fat.'' In a separate study conducted at Purdue University in Indiana, CLA was found to help improve insulin levels in about two-thirds of diabetic patients, and moderately reduced the blood glucose level and triglyceride levels, suggesting that it may help to maintain normal insulin levels.

CLA has been the subject of a variety of research in the past several years, and findings also suggest that some of the other benefits of CLA include the following:

May increase metabolic rate -- This would obviously be a positive benefit for promoting normal thyroid function.
May help decrease abdominal fat.
May enhance muscle growth -- Muscle burns fat, which also contributes to increased metabolism, which is useful in weight loss and management.
Helps maintain normal cholesterol and triglyceride levels.
Helps maintain normal insulin levels.
Helps make it easier to control weight.
Enhances immune system -- Enhancing the immune system's ability to function properly is a positive benefit.
If you're interested in taking CLA to help with weight loss, keep in mind that it's not a magic, and you will need to start a program of diet and exercise in order to successfully lose weight and keep it off.

Wiki's version:


http://en.wikipedia.org/wiki/Conjugated_linoleic_acid


Conjugated linoleic acid (CLA) is a mixture of positional and geometric isomers of linoleic acid, which is found preferentially in dairy products and meat. Preliminary studies indicate that CLA is a powerful antioxidant with an effective range of 0.1-1% in the diet.

Conjugated linoleic acid is unique because it is present in food from animal sources, and its antioxidant efficacy is expressed at concentrations close to human consumption levels.

Conjugated linoleic acid (CLA) refers to a family of many isomers of linoleic acid (at least 13 are reported), which are found primarily in the meat and dairy products of ruminants. As implied by the name, the double bonds of CLAs are conjugated.
Conjugated linoleic acid is a trans fat, though some researchers claim that it is not harmful in the same fashion as other trans fatty acids, but rather is beneficial.[citation needed] CLA is a conjugated system, and in the United States, trans linkages in a conjugated system are not counted as trans fat for the purposes of nutritional regulations and labeling. Unlike most trans fatty acids found in the human diet, CLA occurs naturally, produced by microorganisms in the fore-stomach of ruminants. Non-ruminants, such as humans, may be able to produce some isomers of CLA from non-conjugated ruminant fats. One such example is vaccenic acid, which could be converted to CLA by delta-9-desaturase.[1]
CLA comes in two isomers: the c9,t11 isomer (rumenic acid) which appears responsible for improving muscle growth,[citation needed] and the t10,c12 isomer which primarily prevents lipogenesis (storage of fat in adipose tissue). Most supplements sold in stores contain a 50/50 mix of both isomers. [2]
Various antioxidant and anti-tumor properties have been attributed to CLA, and studies on mice and rats show promising results; however, it is suspected that sufficient concentrations to achieve anti-inflammatory effects within human tissues may not be attainable via oral consumption.[citation needed]
Many studies on CLA in humans show a tendency for reduced body fat[3], particularly abdominal fat, changes in serum total lipids and decreased whole body glucose uptake. The maximum reduction in body fat mass was achieved with a 3.4 g daily dose[4].
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Phosphatidyl serine


http://www.pdrhealth.com/drug_info/nmdrugprofiles/nutsupdrugs/pho_0202.shtml


DESCRIPTION

Phosphatidylserine is a phospholipid that is a structural component of biological membranes of plants, animals and other life forms. Phosphatidylserine was first isolated from brain lipids called cephalins. The major cephalins are phosphatidylserine and phophatidylethanolamine. Another major phospholipid found in egg yolks and soya is phosphatidylcholine, also known, chemically, as lecithin. Phosphatidylserine is also isolated from soya and egg yolks.

Phosphatidylserine is made up of a glycerophosphate skeleton linked to two fatty acid molecules and the amino acid L-serine. It is an amphiphilic molecule because it is made up of the lipophilic fatty acid tails on one side and the hydrophilic head group containing phosphate and serine on the other side of the molecule. Phosphatidylserine is located in the internal layers of biologic membranes, facing the cytoplasm with its polar head group. In animal tissues, phosphatidylserine is formed from phosphatidylethanolamine by exchange of the ethanolamine head for L-serine. Phosphatidylethanolamine itself is synthesized from diacylglycerol and CDP-ethanolamine.

Phosphatidylserine is known chemically as 1,2-diacyl-sn-glycerol-(3)-L-phosphoserine. It is abbreviated as Ptd Ser, Acyl2 Gro PSer and PS. Most commonly, it is called phosphatidylserine or PS. It has the following chemical structure:


Phosphatidylserine

The fatty acid composition of phosphatidylserine derived from bovine brain and soya lecithin differ. Phosphatidylserine from soya lecithin contains mainly polyunsatured fatty acids, while phosphatidylserine derived from bovine brain contains mainly saturated and monounsaturated fatty acids, as well as some docosahexaenoic acid.

Phosphatidylserine is involved in signal transduction activity as well as being a basic structural component of biologic membranes.

ACTIONS AND PHARMACOLOGY

ACTIONS

Supplemental phosphatidylserine may have cognition enhancing activity.

MECHANISMS OF ACTION

Since the action of phosphatidylserine has not been established, any discussion of the mechanism of action is speculative. However, some findings from animal studies are of interest. Cholinergic hypofunction is thought to account in part for the cognitive deficits found in Alzheimer's disease. The most commonly used drugs for the treatment of Alzheimer's disease are reversible acetylcholinesterase inhibitors. The rationale of these drugs is to increase acetylcholine levels in the brains of Alzheimer's patients, and they may be somewhat effective in some cases. Animal studies indicate that phosphatidylserine restores acetylcholine release in aging rats by maintaining an adequate supply of the molecule and is able to increase the availability of endogenous choline for de novo acetylcholine synthesis.

The hippocampus of the brain is believed to be important for cognitive processes and is affected in those with Alzheimer's disease. The dendritic spines of pyramidal cells, the post-synaptic target of the excitatory input to the hippocampus, have been proposed as a substrate for information storage. Age-dependent dendritic spine loss in pyramidal neurons has been reported in the human brain, and the extent of synaptic loss appears to correlate with the degree of cognitive impairment. Rat experiments indicate that phosphatidylserine treatment prevents the age-related reduction in dendritic spine density in rat hippocampus. Protein kinase C facilitation of acetylcholine release has been reported in rats. Phosphatidylserine was found to restore protein kinase C activity in aging rats. Stimulation of calcium uptake by brain synaptosomes and activation of protein kinase C are yet other speculative mechanisms of phosphatidylserine's putative cognition-enhancing action.

PHARMACOKINETICS

Pharmacokinetic studies of phosphatidylserine have been performed in rats. Little is known of the pharmacokinetics of oral phosphatidylserine in humans. In rats, it appears that there is extensive digestion of phosphatidylserine in the small intestine, producing, among other things, lysophosphatidylserine, a substance that contains only one fatty acid, and phosphatidylethanolamine.

Following absorption, lysophosphatidylserine is metabolized in intestinal mucosa cells, and its metabolites, which include some phosphatidylserine, enter the lymphatics draining the small intestine. It appears that only a small fraction of ingested phosphatidylserine reaches the systemic circulation as part of the phospholipid pool. The amount that reaches the brain, after either intraperitoneal injection or oral administration, is very small. Most of the behavioral and neurochemical effects noted in animal studies have been observed only after repeated intraperitoneal and oral phosphatidylserine dosing.

INDICATIONS AND USAGE

Phosphatidylserine has demonstrated some usefulness in treating cognitive impairment, including Alzheimer's disease, age-associated memory impairment and some non-Alzheimer's dementias. More research is needed before phosphatidylserine can be indicated for immune enhancement or for reduction of exercise stress.

RESEARCH SUMMARY

Several double-blind studies suggest that phosphatidylserine can help maintain cognitive function in older individuals and may be able to improve memory and learning skill in some. These results, while encouraging, are not, to date, dramatic.

Various animal experiments have also demonstrated some benefits including stimulation of brain catechecholaminergic turnover and increased acetylcholine output from the cerebral cortex of adult and old rats, as well as enhanced neurotransmitter and central nervous system signal transduction. There is evidence that phosphatidylserine can help maintain the hippocampal dendritic spine population of aging rats. It has been suggested that these spines serve as a substrate for information storage. There are several studies demonstrating improved cognitive function in several animal models.

In the largest multicenter study to date of phosphatidylserine and Alzheimer's disease, 142 subjects aged 40 to 80 were given 200 milligrams of phosphatidylserine per day or placebo over a three-month period. Those treated with phosphatidylserine exhibited improvement on several items on the scales normally used to assess Alzheimer's status. The differences between placebo and experimental groups were small but statistically significant. Researchers directing a smaller study, also achieving statistical significance with respect to several measures, characterized the therapeutic effects of phosphatidylserine in their Alzheimer's subjects as "mild."

Phosphatidylserine has also shown some efficacy in some non-Alzheimer's dementias, in age-associated memory impairment and general mental deterioration. More clinical trials need to be conducted before anything conclusive can be said about phosphatidylserine in the treatment of cognition impairment. But, given the results to date and the fact that there are so few side effects associated with phosphatidylserine and so few treatment options for Alzheimer's disease, one research group concludes that "the therapeutic possibilities offered by phosphatidylserine should not be dismissed." There are some animal studies demonstrating positive immunomodulatory effects but, as yet, no human studies showing similar effects. There is preliminary research indicating that phosphatidylserine, at doses of 400 to 800 milligrams per day, can inhibit exercise-induced increases in cortisol.

CONTRAINDICATIONS, PRECAUTION, ADVERSE REACTIONS

CONTRAINDICATIONS

Phosphatidylserine supplementation is contraindicated in those hypersensitive to any component of the preparation.

PRECAUTIONS

Because of lack of long-term safety studies, phosphatidylserine should be avoided by children, pregnant women and nursing mothers. Those with the antiphospholipid-antibody syndrome should exercise caution in the use of phosphatidylserine and only take it under medical supervision and monitoring.

ADVERSE REACTIONS

Occasional gastrointestinal side effects, such as nausea and indigestion, are reported.

INTERACTIONS

There are no reported drug, nutritional supplement, food or herb interactions with phosphatidylserine.

OVERDOSAGE

There are no reports of overdosage.

LD50 in rats is more than 5g/kg, and in rabbits is more than 2g/kg.

DOSAGE AND ADMINISTRATION

Phosphatidylserine supplements derived from both bovine brain and from soya lecithin are available. Phosphatidylserine derived from soya lecithin undergoes an enzymatic process that converts phosphatidylcholine to phosphatidylserine. Because of the hypothetical possibility of bovine spongiform encephalopathy, the soya-derived phosphatidylserine is preferred. Typical doses are 100 milligrams three time daily.

HOW SUPPLIED

Capsules — 50 mg, 100 mg, 500 mg Tweet