antioxidants and endometriosis

just a couple of links~

http://www.doctoryourself.com/endometriosis.html


Selenium
In cattle, endometriosis can be due to selenium deficiency. Since your cow can't give you any milk without having a baby first, dairymen well know to supplement all cows' feed with selenium. This is usually accomplished with a multimineral tablet the size of a microwave oven. Okay, it's a mineral-fortified salt block (after all, salt IS a mineral...) that can cows lick anytime they want.

Human females should do as well, but they don't.

We go out of our way to supply selenium to cattle, particularly in geographical regions that have selenium-poor soil (New York state is just one of these). Farmers simply must have healthy, fertile, easy-to-conceive, happily-pregnant, uncomplicated-delivering Bossys by the herd. The economics are obvious: farmer's cannot afford otherwise; a herd with endometriosis would be bankruptcy on the hoof.

Women with endometriosis, on the other hand, mean economic success for all the many, many systems we have to capitalize on their sickness. Doctors, nurses, support staff, surgeons, hospitals, administrators, HMO's, insurance companies, pharmaceutical manufacturers, drug salespeople, and lots of others' jobs depend on people being unwell. The uterus has always been irresistible to money-medicine, and, with an estimated half a million unnecessary hysterectomies annually, I would say that little has changed.

The secret to endometriosis is to see it for what it is: an end result of malnutrition.

Farmers see this. Physicians do not.

Cows are raw food vegetarians who obtain their minerals from grains, leafy greens, and smart dairymen who provide mineral supplements preventively. Physicians and their ilk try to treat endometriosis 1) after it has occurred and 2) with drugs. Endometriosis is not due to a drug deficiency.

The secret to this disease is found in the barnyard.

Selenium is probably important in stopping endometriosis because this important trace mineral works so closely with vitamin E. Vitamin E has been known to ensure that animals have healthy uterine linings since the 1930's. There is a research trail on this as long as your arm. Therefore, supplementing the diet of a human female with (natural) vitamin E, 400 to 1,000 IU daily (Balch, 1990, p 167), plus 100-200 micrograms (mcg) of selenium, is a good move.

Folate (folic acid)
I suspect folate deficiency as a cause of endometriosis. To some extent I base this opinion, once again, on cows. And I repeat that cows are vegetarians. Raw food vegetarians.

Folate, once known as vitamin B - 9, is named after the dark green leafy vegetables it was first extracted from. "Folium" is Latin for leaf. Folic acid contains three parts: pteroic acid glutamic acid, and para-amino benzoic acid (PABA). Folate is an important coenzyme in your body which helps to move carbon units about, and is necessary for the synthesis of nitrogen-containing purines and pyrimidines, which are essential for the synthesis of nucleotides... which make up your RNA and DNA. Folate is also necessary for making the heme (the iron-containing, nonprotein part of hemoglobin) for your red blood cells

Too little folate causes nutritional megoblastic anemia (that's large, immature red blood cells that can't carry oxygen well). This is especially important during growth situations, such as pregnancy, infancy and childhood.

Cows get plenty of folate because they eat plenty of foliage (green leafy stuff, like grass). They are also blissfully free from a silent folate-stealer: the birth control pill. Oral contraceptives dramatically increase (at least double) the need for folic acid in women. Disease in general increases need for folic acid.

Adolescent girls in particular are likely to have folic acid deficiencies (Williams, 6th, p 245) Why? Because food sources of folate are often quite unpopular. They are:

1. green leafy vegetables (Teens LOVE these. Not.)
2. organ meats ("Awesome!)
3. asparagus ("McSparagus! My favorite fast food!")

It is likely that, during the pre-adult growth period when they need it most, teenagers are not getting adequate dietary folic acid. Female teenagers reaching menarche (beginning of menstruation) are therefore malnourished. Folate undernutrition is probably a factor in endometriosis.

Other nutrients that James F. Balch, M.D. recommends for endometriosis include vitamin C in quantity, the vitamin B-complex, essential fatty acids (found in lecithin or primrose oil), iron, iodine, calcium, and magnesium (Balch, p 167). His concise discussion of endometriosis (pages 165-168) includes a discussion a theory that endometriosis may actually be an "unrecognized congenital birth defect." If this is true, surgical correction may be warranted. But likewise if true, prevention must be emphasized. Since there is no question whatsoever that malnutrition causes birth defects, dietary revision makes sense. What is good for heifers is good for humans. I vote for a bovine diet.

Raspberry leaves are rich in magnesium and have a long tradition of uterine usefulness. I have seen raspberry leaf tea reduce pregnancy problems and delivery times in humans. We fed piles of raspberry leaves to our rabbit, who rewarded us with ten young practically while our backs were turned. This is a large litter even for a rabbit.

Pregnancy and endometriosis are not in a cart-and-horse relationship: it is not known for certain which one influences the other. But I think the less sure we are, the more we should look to nature for our examples.

Deficient intake of antioxidant molecules is related to peripheral and peritoneal oxidative stress in endometriosis

Deficient intake of antioxidant molecules is related to peripheral and peritoneal oxidative stress in endometriosis

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http://www.endometriosis.org/research-antioxidants.html



Researchers in Mexico have evaluated the antioxidant intake and the lipoperoxidation status in plasma and peritoneal fluid of women with endometriosis.

They demonstrated that there was an inverse correlation with the pathology intensity depending on the antioxidant intake in women with endometriosis. As endometriosis severity intensifies there is less of an intake of antioxidants. They also demonstrated a positive association between the pathology development and the lipoperoxidation status.

The study was carried out in 48 women with endometriosis obtained at the Sterility Clinic from the National Institute of Perinatology. The antioxidant intake was evaluated by applying the Questionnaire Assessment of Antioxidants and Retinol Intakes in Mexican women validated by the National Institute of Public Health. The lipoperoxidation status was determined in plasma and peritoneal fluid with the malondyaldehyde method. Results were analysed by ANOVA of Kruskal-Wallis.

The researchers showed that antioxidant intake (vitamin C, vitamin E, selenium and zinc) in the women with endometriosis showed a significative statistical difference when compared with the control group, including the total group and those stratified in pathology stages. The lipoperoxidation status in plasma and peritoneal fluid of women with endometriosis did not show statistical difference versus healthy women. When data was stratified according to the severity of pathology, the percentage of lipoperoxidation status increased in plasma in the severe grade of endometriosis and in peritoneal fluid in the moderate and severe grades.


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