Joyce, I am not a big fan of sodium bicarbonate and maple syrup to begin with. Other than cancers that occur from the mouth through the digestive tract and on to the anus, there is really no way for the sodium bicarbonate to reach the site of the cancer. It might help lower pH a bit, but the way human digestion works there is simply no way for a mixture of maple syrup and baking soda to make it through the digestion system into the blood stream and to tumors such as those found in the uterus and cervix. To think otherwise is pretty much in the category of uban myth. It is my further opinion that the biggest proponent for sodium bicarbonate against cancers, Dr. Tulio Simoncini, is pretty much a fraud and charlatan.
As one astute observer opined, baking soda will also remove corrosion on battery terminals - but it won't get there to do the job if you put it in your gas tank.
If it appears to be working for you for whatever reason, that is fine. I would submit that the anti-cancer protocol I suggest would work far better - and nowhere in it will you find a recommendation for sodium bicarbonate or maple syrup.
All the best,
Tony
Sugar spells candida? Well, candida con lead to cancer, but it does not spell cancer from the standpoint of cancer being a fungus - which is a bogus theory promoted by a bogus cancer authority, imo.
Slavek, while I can't say for certain whether or not ascorbate acid "drags" some sodium with it, the whole mythology about maple syrup and baking soda is the stuff of the worst sort of urban legend and covoluted logic. I think it began in large part due to the overhype by Tulio Simoncini (speaking of bogus cancer authorities and bogus theories) about his limited and overplayed results of directly injecting sodium bicarbonate into tumors and/or intravenously. I have virtually no respect for him based on what I have learned about his bogus testimonials, over-hyped results and apparent deaths which were evidently due to his procedures as well as his lack of proper treatment and monitioring of his patients.
When it comes to baking soda and maple syrup, the idea sounds like something some beer drinking Bubbas would come up with - that somehow maple syrup would perform a protective coating around baking soda and that this compound would somehow miraculously makeit intact through the digestive system and into the blood stream (and impossibility) and then make it to the cancer tumor site where the cancer cells would be fooled into taking this concocted compound inside the cancer cells where the baking soda would then be released and destroy the cancer cells. Utter hogwash and quite impossible. Now, there might be some benefits derived from the baking soda which offset feeding the sugars in maple syrup to cancer cells, but not in the way it has been described.
I am with you when it comes to sugar and cancer specifically and refiined sugar and health in general and refer you to this article by PhD Nancy Appleton which gives a very good idea why the WHO had identified refined sugar as the number one cause of health problems in the world today:
146 Reasons Why Sugar Is Ruining Your Health
Nancy Appleton, Ph.D.
1. Sugar can suppress the immune system.
2. Sugar upsets the mineral relationships in the body.
3. Sugar can cause hyperactivity, anxiety, difficulty concentrating, and crankiness in children.
4. Sugar can produce a significant rise in triglycerides.
5. Sugar contributes to the reduction in defense against bacterial infection (infectious diseases).
6. Sugar causes a loss of tissue elasticity and function, the more sugar you eat the more elasticity and function you loose.
7. Sugar reduces high-density lipoproteins.
8. Sugar leads to chromium deficiency.
9. Sugar leads to cancer of the ovaries.
10. Sugar can increase fasting levels of glucose.
11. Sugar causes copper deficiency.
12. Sugar interferes with absorption of calcium and magnesium.
13. Sugar may make eyes more vulnerable to age-related macular degeneration.
14. Sugar raises the level of a neurotransmitters: dopamine, serotonin, and norepinephrine.
15. Sugar can cause hypoglycemia.
16. Sugar can produce an acidic digestive tract.
17. Sugar can cause a rapid rise of adrenaline levels in children.
18. Sugar malabsorption is frequent in patients with functional bowel disease.
19. Sugar can cause premature aging.
20. Sugar can lead to alcoholism.
21. Sugar can cause tooth decay.
22. Sugar contributes to obesity
23. High intake of sugar increases the risk of Crohn's disease, and ulcerative colitis.
24. Sugar can cause changes frequently found in person with gastric or duodenal ulcers.
25. Sugar can cause arthritis.
26. Sugar can cause asthma.
27. Sugar greatly assists the uncontrolled growth of Candida Albicans (yeast infections).
28. Sugar can cause gallstones.
29. Sugar can cause heart disease.
30. Sugar can cause appendicitis.
31. Sugar can cause hemorrhoids.
32. Sugar can cause varicose veins.
33. Sugar can elevate glucose and insulin responses in oral contraceptive users.
34. Sugar can lead to periodontal disease.
35. Sugar can contribute to osteoporosis.
36. Sugar contributes to saliva acidity.
37. Sugar can cause a decrease in insulin sensitivity.
38. Sugar can lower the amount of Vitamin E (alpha-Tocopherol) in the blood.
39. Sugar can decrease growth hormone.
40. Sugar can increase cholesterol.
41. Sugar can increase the systolic blood pressure.
42. High sugar intake increases advanced glycation end products (AGEs)(Sugar bound non-enzymatically to protein)
43. Sugar can interfere with the absorption of protein.
44. Sugar causes food allergies.
45. Sugar can contribute to diabetes.
46. Sugar can cause toxemia during pregnancy.
47. Sugar can contribute to eczema in children.
48. Sugar can cause cardiovascular disease.
49. Sugar can impair the structure of DNA
50. Sugar can change the structure of protein.
51. Sugar can make our skin age by changing the structure of collagen.
52. Sugar can cause cataracts.
53. Sugar can cause emphysema.
54. Sugar can cause atherosclerosis.
55. Sugar can promote an elevation of low-density lipoproteins (LDL).
56. High sugar intake can impair the physiological homeostasis of many systems in the body.
57. Sugar lowers the enzymes ability to function.
58. Sugar intake is higher in people with Parkinson’s disease.
59. Sugar can increase the size of the liver by making the liver cells divide.
60. Sugar can increase the amount of liver fat.
61. Sugar can increase kidney size and produce pathological changes in the kidney.
62. Sugar can damage the pancreas.
63. Sugar can increase the body's fluid retention.
64. Sugar is enemy #1 of the bowel movement.
65. Sugar can cause myopia (nearsightedness).
66. Sugar can compromise the lining of the capillaries.
67. Sugar can make the tendons more brittle.
68. Sugar can cause headaches, including migraine.
69. Sugar plays a role in pancreatic cancer in women.
70. Sugar can adversely affect school children's grades and cause learning disorders.
71. Sugar can cause depression.
72. Sugar increases the risk of gastric cancer.
73. Sugar and cause dyspepsia (indigestion).
74. Sugar can increase your risk of getting gout.
75. Sugar can increase the levels of glucose in an oral glucose tolerance test over the ingestion of complex carbohydrates.
76. Sugar can increase the insulin responses in humans consuming high-sugar diets compared to low-sugar diets.
77. A diet high in refined sugar reduces learning capacity.
78. Sugar can cause less effective functioning of two blood proteins, albumin, and lipoproteins, which may reduce the body’s ability to handle fat and cholesterol.
79. Sugar can contribute to Alzheimer’s disease.
80. Sugar can cause platelet adhesiveness.
81. Sugar can cause hormonal imbalance; some hormones become under active and others become overactive.
82. Sugar can lead to the formation of kidney stones.
83. Diets high in sugar can cause free radicals and oxidative stress.
84. High sugar diet can lead to biliary tract cancer.
85. High sugar consumption of pregnant adolescents is associated with a twofold-increased risk for delivering a small-for-gestational-age (SGA) infant.
86. High sugar consumption can lead to substantial decrease in gestation duration among adolescents.
87. Sugar slows food's travel time through the gastrointestinal tract.
88. Sugar increases the concentration of bile acids in stools and bacterial enzymes in the colon. This can modify bile to produce cancer-causing compounds and colon cancer.
89. Sugar increases estradiol (the most potent form of naturally occurring estrogen) in men.
90. Sugar combines with and destroys phosphatase, an enzyme, which makes the process of digestion more difficult.
91. Sugar can be a risk factor of gallbladder cancer.
92. Sugar is an addictive substance.
93. Sugar can be intoxicating, similar to alcohol.
94. Sugar can exacerbate PMS.
95. Sugar given to premature babies can affect the amount of carbon dioxide they produce.
96. Decrease in sugar intake can increase emotional stability.
97. The rapid absorption of sugar promotes excessive food intake in obese subjects.
98. Sugar can worsen the symptoms of children with attention deficit hyperactivity disorder (ADHD).
99. Sugar adversely affects urinary electrolyte composition.
100. Sugar can slow down the ability of the adrenal glands to function.
101.. I.Vs (intravenous feedings) of sugar water can cut off oxygen to the brain.
102. High sucrose intake could be an important risk factor in lung cancer.
103. Sugar increases the risk of polio.
104. High sugar intake can cause epileptic seizures.
105. Sugar causes high blood pressure in obese people.
106. In Intensive Care Units, limiting sugar saves lives.
107. Sugar may induce cell death.
108. Sugar can increase the amount of food that you eat.
109. In juvenile rehabilitation camps, when children were put on a low sugar diet, there was a 44% drop in antisocial behavior.
110. Sugar can lead to prostrate cancer.
111. Sugar dehydrates newborns.
112. Sugar can cause low birth weight babies.
113. Greater consumption of refined sugar is associated with a worse outcome of schizophrenia
114. Sugar can raise homocysteine levels in the blood stream.
115. Sweet food items increase the risk of breast cancer.
116. Sugar is a risk factor in cancer of the small intestine.
117. Sugar may cause laryngeal cancer.
118. Sugar induces salt and water retention.
119. Sugar may contribute to mild memory loss.
120. The more sodas a 10 year old child consumes, the less milk.
121. Sugar can increase the total amount of food consumed.
122. Exposing a newborn to sugar results in a heightened preference for sucrose relative to water at 6 months and 2 years of age.
123. Sugar causes constipation.
124. Sugar causes varicose veins.
125. Sugar can cause brain decay in prediabetic and diabetic women.
126. Sugar can increase the risk of stomach cancer.
127. Sugar can cause metabolic syndrome.
128. Sugar ingestion by pregnant women increases neural tube defects in embryos.
129. Sugar can be a factor in asthma.
130. The higher the sugar consumption the more chances of getting irritable bowel syndrome.
131. Sugar can affect the brain’s ability to deal with rewards and consequences.
132. Sugar can cause cancer of the rectum.
133. Sugar can cause endometrial cancer.
134. Sugar can cause renal (kidney) cell carcinoma.
135. Sugar can cause liver tumors.
136. Sugar can increase inflammatory markers in the blood stream of overweight people.
137. Sugar can lower Vitamin E levels in the blood stream.
138. Sugar can increase your appetite for all food.
139. Sugar plays a role in the etiology and the continuation of acne.
140. Too much sugar can kill your sex life.
141. Sugar saps school performance in children.
142. Sugar can cause fatigue, moodiness, nervousness and depression.
143. Sugar is common choice of obese individuals.
144. A linear decrease in the intake of many essential nutrients is associated with increasing total sugar intake.
145. High fructose consumption has been linked to liver disease.
146. Sugar adds to the risk of bladder cancer.
--------------------------------------------
1. Sanchez, A., et al. "Role of Sugars in Human Neutrophilic Phagocytosis," Am J Clin Nutr. Nov 1973;261:1180-1184.
Bernstein, J., et al. "Depression of Lymphocyte Transformation Following Oral Glucose Ingestion." Am J Clin Nutr. 1997;30:613.
2. Couzy, F., et al."Nutritional Implications of the Interaction Minerals," Progressive Food and Nutrition Science. 17;1933:65-87.
3. Goldman, J., et al. "Behavioral Effects of Sucrose on Preschool Children." J Abnormal Child Psychol. 1986;14(4):565-577.
4. Scanto, S. and Yudkin, J. "The Effect of Dietary Sucrose on Blood Lipids, Serum Insulin, Platelet Adhesiveness and Body Weight in Human Volunteers." J Postgrad Med. 1969;45:602-607.
5. Ringsdorf, W., Cheraskin, E. and Ramsay R. "Sucrose,Neutrophilic Phagocytosis and Resistance to Disease," Dental Surv. 1976;52(12):46-48.
6. Cerami, A., et al. "Glucose and Aging." Scientific American. May 1987:90.
Lee, A. T. and Cerami, A. "The Role of Glycation in Aging." Ann N Y Acad Sci. 663:63-67.
7. Albrink, M. and Ullrich I. H. "Interaction of Dietary Sucrose and Fiber on Serum Lipids in Healthy Young Men Fed High Carbohydrate Diets." Am J Clin Nutr. 1986;43:419-428.
Pamplona, R., et al. “Mechanisms of Glycation in Atherogenesis.” Med Hypotheses. Mar 1993;40(3):174-81.
8. Kozlovsky, A., et al. "Effects of Diets High in Simple Sugars on Urinary Chromium Losses." Metabolism. June 1986;35:515-518.
9. Takahashi, E., Tohoku University School of Medicine, Wholistic Health Digest. October 1982:41.
10. Kelsay, J., et al. "Diets High in Glucose or Sucrose and Young Women." Am J Clin Nutr. 1974;27:926-936.
Thomas, B. J., et al. “Relation of Habitual Diet to Fasting Plasma Insulin Concentration and the Insulin Response to Oral Glucose,” Hum Nutr Clin Nutr. 1983; 36C(1):49_51.
11. Fields, M.., et al. "Effect of Copper Deficiency on Metabolism and Mortality in Rats Fed Sucrose or Starch Diets," Am J Clin Nutr. 1983;113:1335-1345.
12. Lemann, J. "Evidence that Glucose Ingestion Inhibits Net Renal Tubular Reabsorption of Calcium and Magnesium." Am J Clin Nutr. 1976 ;70:236-245.
13. Chiu, C. Am J Clin Nutr. July 2007;86:180-188
14. "Sugar, White Flour Withdrawal Produces Chemical Response." The Addiction Letter .Jul 1992:4.
15. Dufty, William. Sugar Blues. (New York:Warner Books, 1975).
16. Ibid.
17. Jones, T. W., et al. “Enhanced Adrenomedullary Response and Increased Susceptibility to Neuroglygopenia: Mechanisms Underlying the Adverse Effect of Sugar Ingestion in Children.” J Pediatrics. Feb 1995;126:171-7.
18. Ibid.
19. Lee, A. T.and Cerami A. "The Role of Glycation in Aging." Annals of the New York Academy of Science. 1992;663:63-70.
20. Abrahamson, E. and Peget, A. Body, Mind and Sugar. (New York:Avon,1977.)
21. Glinsmann, W., et al. “Evaluation of Health Aspects of Sugar Contained in Carbohydrate Sweeteners.” F. D. A. Report of Sugars Task Force. 1986:39.
Makinen K.K.,et al. “A Descriptive Report of the Effects of a 16_month Xylitol Chewing_Gum Programme Subsequent to a 40_Month Sucrose Gum Programme.” Caries Research. 1998; 32(2)107-12.
Riva Touger-Decker and Cor van Loveren, “Sugars and Dental Caries.”
Am. J. Clin.Nutr. Oct 2003; 78:881-892.
22. Keen, H., et al. "Nutrient Intake, Adiposity, and Diabetes." Brit Med J. 1989; 1: 655-658.
23. Tragnone, A. et al. “Dietary Habits as Risk Factors for Inflammatory Bowel Disease.” Eur J Gastroenterol Hepatol. Jan 1995;7(1):47-51.
24. Yudkin, J. Sweet and Dangerous. (New York;Bantam Books:1974), 129.
25. Darlington, L., Ramsey, et al. "Placebo-Controlled, Blind Study of Dietary Manipulation Therapy in Rheumatoid Arthritis," Lancet. Feb 1986;8475(1):236-238.
26. Powers, L. "Sensitivity: You React to What You Eat." Los Angeles Times. Feb. 12, 1985.
Cheng, J., et al. “Preliminary Clinical Study on the Correlation Between Allergic Rhinitis and Food Factors.” Lin Chuang Er Bi Yan Hou Ke Za Zhi Aug 2002;16(8):393-396.
27. Crook, W. J. The Yeast Connection. (TN:Professional Books, 1984).
28. Heaton, K. "The Sweet Road to Gallstones." Brit Med J. Apr 14, 1984; 288:1103-1104.
Misciagna, G., et al. Am J Clin Nutr. 1999;69:120-126.
29. Yudkin, J. "Sugar Consumption and Myocardial Infarction." Lancet. Feb 6, 1971;1(7693):296-297.
Chess DJ, et al. “Deleterious Effects of Sugar and Protective Effects of Starch on Cardiac Remodeling, Contractile Dysfunction, and Mortality in Response to Pressure Overload.” Am J Physiol Heart Circ Physiol. Sept. 2007;293(3):H1853-H1860
30. Cleave, T. The Saccharine Disease. (New Canaan, CT: Keats Publishing, 1974).
31. op. cit.
32. Cleave, T. and Campbell, G. Diabetes, Coronary Thrombosis and the Saccharine Disease. (Bristol, England, John Wright and Sons, 1960).
33. Behall, K. "Influence of Estrogen Content of Oral Contraceptives and Consumption of Sucrose on Blood Parameters." Disease Abstracts International. 1982;431-437.
34. Glinsmann, W., et al. “Evaluation of Health Aspects of Sugar Contained in Carbohydrate Sweeteners.” F. D. A. Report of Sugars Task Force. 1986;39:36_38.
35. Tjäderhane, L. and Larmas, M. “A High Sucrose Diet Decreases the Mechanical Strength of Bones in Growing Rats.” Am J Clin Nutr. 1998:128:1807-1810.
36. Wilson R.F. and Ashley F.P. “The Effects of Experimental Variations in Dietary Sugar Intake and Oral hygiene on the Biochemical Composition and pH of Free Smooth-surface and Approximal Plaque.” J Dent Res. June 1988;67(6):949-953
37. Beck-Nielsen H., et al. “Effects of Diet on the Cellular Insulin Binding and the Insulin Sensitivity in Young Healthy Subjects." Diabetes. 1978;15:289-296.
38. Mohanty P. et al. “Glucose Challenge Stimulates Reactive Oxygen Species (ROS) Generation by Leucocytes.” J Clin Endocrin Metabol. Aug 2000; 85(8):2970-2973.
39. Gardner, L. and Reiser, S. "Effects of Dietary Carbohydrate on Fasting Levels of Human Growth Hormone and Cortisol." Proc Soc Exp Biol Med. 1982;169:36-40.
40. Ma Y. et al. “Association Between Carbohydrate Intake and Serum Lipids.” J Am Coll Nutr. Apr 2006;25(2):155-163
41. Preuss, H. G. “Sugar-Induced Blood Pressure Elevations Over the Lifespan of Three Substrains of Wistar Rats.” J Am Coll of Nutr. 1998;17(1) 36-37.
42. Furth, A. and Harding, J. "Why Sugar Is Bad For You." New Scientist. Sep 23, 1989;44.
43. Lee AT, Cerami A. “Role of Glycation in Aging.” Ann N Y Acad Sci. Nov 21, 1992 ;663:63-70.
44. Appleton, N. Lick the Sugar Habit. (New York: Avery Penguin Putnam:1988).
45. Henriksen H. B. and, Kolset S.O. Tidsskr Nor Laegeforen. Sep 6, 2007;127(17):2259-62.
46. Cleave, T.: The Saccharine Disease. (New Canaan Ct: Keats Publishing, Inc., 1974).131.
47. Ibid. 132.
48. Vaccaro O., et al. “Relationship of Postload Plasma Glucose to Mortality with 19 Year Follow-up.” Diabetes Care. Oct 15,1992;10:328-334.
Tominaga, M., et al, “Impaired Glucose Tolerance Is a Risk Factor for Cardiovascular Disease, but Not Fasting Glucose.” Diabetes Care. 1999:2(6):920-924.
49. Lee, A. T. and Cerami, A. "Modifications of Proteins and Nucleic Acids by Reducing Sugars: Possible Role in Aging." Handbook of the Biology of Aging. (New York: Academic Press, 1990.).
50. Monnier, V. M. "Nonenzymatic Glycosylation, the Maillard Reaction and the Aging Process." J Gerontol. 1990:45(4 ):105-110.
51. Dyer, D. G., et al. "Accumulation of Maillard Reaction Products in Skin Collagen in Diabetes and Aging." J Clin Invest. 1993:93(6):421-422.
52. Veromann, S. et al. ”Dietary Sugar and Salt Represent Real Risk Factors for Cataract Development.” Ophthalmologica. Jul-Aug 2003 ;217(4):302-307.
53. Monnier, V. M. "Nonenzymatic Glycosylation, the Maillard Reaction and the Aging Process." J Gerontol. 1990:45(4):105-110.
54. Schmidt A.M. et al. “Activation of Receptor for Advanced Glycation End Products: a Mechanism for Chronic Vascular Dysfunction in Diabetic Vasculopathy and Atherosclerosis.” Circ Res. Mar 1999 19;84(5):489-97.
55. Lewis, G. F. and Steiner, G. “Acute Effects of Insulin in the Control of VLDL Production in Humans. Implications for The Insulin-resistant State.” Diabetes Care. Apr 1996;19(4):390-3
R. Pamplona, M. .J., et al. "Mechanisms of Glycation in Atherogenesis." Med Hypotheses. 1990;40:174-181.
56. Ceriello, A. “Oxidative Stress and Glycemic Regulation.” Metabolism. Feb 2000;49(2 Suppl 1):27-29.
57. Appleton, N. Lick the Sugar Habit. (New York: Avery Penguin Putnam, 1988).
58. Hellenbrand, W. ”Diet and Parkinson's Disease. A Possible Role for the Past Intake of Specific Nutrients. Results from a Self-administered Food-frequency Questionnaire in a Case-control Study.” Neurology. Sep 1996;47(3):644-650
Cerami, A., et al. "Glucose and Aging." Scientific American. May 1987: 90.
59. Goulart, F. S. "Are You Sugar Smart?" American Fitness. Mar-Apr 1991: 34-38.
60. Scribner, K.B. et al. “Hepatic Steatosis and Increased Adiposity in Mice Consuming Rapidly vs. Slowly Absorbed Carbohydrate.” Obesity. 2007;15:2190-2199.
61. Yudkin, J., Kang, S. and Bruckdorfer, K. "Effects of High Dietary Sugar." Brit J Med. Nov 22, 1980;1396.
62. Goulart, F. S. "Are You Sugar Smart?" American Fitness. March-April 1991: 34-38
63. Ibid.
64. Ibid.
65. Ibid.
66. Ibid.
67. Nash, J. "Health Contenders." Essence. Jan 1992-23: 79-81.
68. Grand, E. "Food Allergies and Migraine." Lancet. 1979:1:955-959.
69. Michaud, D. ”Dietary Sugar, Glycemic Load, and Pancreatic Cancer Risk in a Prospective Study.” J Natl Cancer Inst. Sep 4, 2002 ;94(17):1293-300.
70. Schauss, A. Diet, Crime and Delinquency. (Berkley Ca; Parker House, 1981).
71. Peet, M. "International Variations in the Outcome of Schizophrenia and the Prevalence of Depression in Relation to National Dietary Practices: An Ecological Analysis." Brit J Psych. 2004;184:404-408.
72. Cornee, J., et al. "A Case-control Study of Gastric Cancer and Nutritional Factors in Marseille, France," Eur J Epidemiol. 1995;11:55-65.
73. Yudkin, J. Sweet and Dangerous. (New York: Bantam Books,1974) 129.
74. . Choi HK, and Curhan G. “Soft drinks, fructose consumption, and the risk of gout in men: prospective cohort study.” British Medical Journal. Feb. 9,2008;336(7639):309-312.
75. Reiser, S., et al. “Effects of Sugars on Indices on Glucose Tolerance in Humans." Am J Clin Nutr. 1986:43;151-159.
76. Reiser,S., et al. “Effects of Sugars on Indices on Glucose Tolerance in Humans." Am J Clin Nutr. 1986;43:151-159.
77. Molteni, R, et al. “A High-fat, Refined Sugar Diet Reduces Hippocampal Brain-derived Neurotrophic Factor, Neuronal Plasticity, and Learning.” NeuroScience. 2002;112(4):803-814.
78. Monnier, V., “Nonenzymatic Glycosylation, the Maillard Reaction and the Aging Process.” J Gerontol. 1990;45:105-111.
79. Frey, J. “Is There Sugar in the Alzheimer’s Disease?” Annales De Biologie Clinique. 2001; 59 (3):253-257.
80. Yudkin, J. "Metabolic Changes Induced by Sugar in Relation to Coronary Heart Disease and Diabetes." Nutrition and Health. 1987;5(1-2):5-8.
81. Ibid.
82. Blacklock, N. J., "Sucrose and Idiopathic Renal Stone." Nutrition and Health. 1987;5(1-2):9-12.
Curhan, G., et al. “Beverage Use and Risk for Kidney Stones in Women.” Ann Inter Med. 1998:28:534-340.
83. Ceriello, A. “Oxidative Stress and Glycemic Regulation.” Metabolism. Feb 2000;49(2 Suppl 1):27-29.
84. Moerman, C. J., et al. “Dietary Sugar Intake in the Etiology of Biliary Tract Cancer.” Internat J Epidemiol. Apr 1993;2(2):207-214.
85. Lenders, C. M. “Gestational Age and Infant Size at Birth Are Associated with Dietary Intake among Pregnant Adolescents.” Am J Nutr. Jun 1997;1113-1117.
86. Ibid.
87. Bostick, R. M., et al. "Sugar, Meat.and Fat Intake and Non-dietary Risk Factors for Colon Cancer Incidence in Iowa Women." Cancer Causes & Control. 1994:5:38-53.
88. Ibid.
Kruis, W., et al. "Effects of Diets Low and High in Refined Sugars on Gut Transit, Bile Acid Metabolism and Bacterial Fermentation.” Gut. 1991;32:367-370.
Ludwig, D. S., et al. “High Glycemic Index Foods, Overeating, And Obesity.” Pediatrics. Mar 1999;103(3):26-32.
89. Yudkin, J and Eisa, O. “Dietary Sucrose and Oestradiol Concentration in Young Men.” Ann Nutr Metabol. 1988:32(2):53-55.
90. Lee, A. T. and Cerami A. "The Role of Glycation in Aging." Ann N Y Acad Sci. 1992; 663:63-70.
91. Moerman, C. et al."Dietary Sugar Intake in the Etiology of Gallbladder Tract Cancer." Internat J of Epi. Apr 1993; 22(2):207-214.
92. Avena N.M. “Evidence for Sugar Addiction: Behavioral and Nuerochemical Effects of Intermittent, Excessive Sugar Intake.” Neurosci Biobehav Rev. 2008;32(1):20-39.
Colantuoni, C., et al. “Evidence That Intermittent, Excessive Sugar Intake Causes Endogenous Opioid Dependence.” Obes Res. Jun 2002 ;10(6):478-488.
93. Ibid.
94. The Edell Health Letter. Sept 1991;7:1.
95. Sunehag, A. L., et al. “Gluconeogenesis in Very Low Birth Weight Infants Receiving Total Parenteral Nutrition.” Diabetes. 1999 ;48 7991-8000).
96. Christensen L. et al. “Impact of A Dietary Change on Emotional Distress.” J Abnor Psychol. 1985;94(4):565-79.
97. Ludwig, D. S., et al. “High Glycemic Index Foods, Overeating and Obesity.” Pediatrics. Mar1999;103(3):26-32.
98. Girardi, N.L.” Blunted Catecholamine Responses after Glucose Ingestion in Children with Attention Deficit Disorder.” Pediatrics Res. 1995;38:539-542.
Berdonces, J. L. “Attention Deficit and Infantile Hyperactivity.” Rev Enferm. Jan 2001;4(1)11-4
99. Blacklock, N. J. “Sucrose and Idiopathic Renal Stone.” Nutrition and Health. 1987;5(1 & 2):9-17.
100. Lechin, F., et al. “Effects of an Oral Glucose Load on Plasma Neurotransmitters in Humans.” Neurophychobiology. 1992;26(1-2):4-11.
101. Arieff, A. I. “IVs of Sugar Water Can Cut Off Oxygen to the Brain.” Veterans Administration Medical Center in San Francisco. San Jose Mercury; June 12/86.
102. De Stefani, E. “Dietary Sugar and Lung Cancer: a Case Control Study in Uruguay.” Nutr and Cancer. 1998;31(2):132_7.
103. Sandler, B.P. Diet Prevents Polio. (Milwakuee, WI,: The Lee Foundation for Nutritional Research, 1951).
104. Murphy, P. “The Role of Sugar in Epileptic Seizures.” Townsend Letter for Doctors and Patients. May, 2001.
105. Stern, N. & Tuck, M. “Pathogenesis of Hypertension in Diabetes Mellitus.” Diabetes Mellitus, a Fundamental and Clinical Test. 2nd Edition, (Phil. A: Lippincott Williams & Wilkins, 2000)943-957.
106. Christansen, D. “Critical Care: Sugar Limit Saves Lives.” Science News. June 30, 2001;159:404.
107. Donnini, D. et al. “Glucose May Induce Cell Death through a Free Radical-mediated Mechanism.” Biochem Biohhys Res Commun. Feb 15, 1996:219(2):412-417.
108. Levine, A.S, et al. “Sugars and Fats: The Neurobiology of Preference “ Am J Nutr. 2003 133:831S-834S.
109. Schoenthaler, S. “The Los Angeles Probation Department Diet-Behavior Program: Am Empirical Analysis of Six Institutional Settings.” Int J Biosocial Res. 5(2):88-89.
110. Deneo-Pellegrini H,. et al.Foods, “Nutrients and Prostate cancer: a Case-control study in Uruguay.” Br J Cancer. 1999 May;80(3-4):591-7.
111. “Gluconeogenesis in Very Low Birth Weight Infants Receiving Total Parenteral Nutrition.” Diabetes. 1999 Apr;48(4):791-800.
112. Lenders, C. M. “Gestational Age and Infant Size at Birth Are Associated with Dietary Intake Among Pregnant Adolescents.” Am Jf Nutr. 1998;128:807-1810.
113. Peet, M. “International Variations in the Outcome of Schizophrenia and the Prevalence of Depression in Relation to National Dietary Practices: An Ecological Analysis.” Brit J Psychiatry. 2004;184:404-408.
114. Fonseca, V. et al. “Effects of a High-fat-sucrose Diet on Enzymes in Homosysteine Metabolism in the Rat.” Metabolism. 200; 49:736-41.
115. Potischman, N, et.al. “Increased Risk of Early-stage Breast Cancer Related to Consumption of Sweet Foods among Women Less than Age 45 in the United States." Cancer Causes Control. 2002 Dec;13(10):937-46.
116. Negri. E. et al. “Risk Factors for Adenocarcinoma of the Small Intestine.”
Intern J Cancer. 1999:82:I2:171-174.
117. Bosetti, C. et al. “Food Groups and Laryngeal Cancer Risk: A Case-control Study from Italy and Switzerland.” Inter J Cancer. 2002:100(3): 355-358.
118. Shannon, M. “An Empathetic Look at Overweight.” CCL Family Found. Nov-Dec.1993. 20(3):3-5.
119. “Health After 50.” Johns Hopkins Med Letter. May 1994.
120.. Rajeshwari, R. et al.”Secular Trends in Children’s Sweetened-beverage Consumption (1973 to 1994): The Bogalusa Heart Study.” J AM Diet Assoc. Feb 205;105(2):208-214.
121. Levine, A.S. et al. "Sugars and Fats: The Neurobiology of Preference." Am J Nutr, 2003;133:831S-834S.
122. Booth, D.A.M. et al. “Sweetness and Food Selection: Measurement of Sweeteners’ Effects on Acceptance.” Sweetness. Dobbing, J., Ed., (London:Springer-Verlag, 1987).
123. Cleve, T.L On the Causation of Varicose Veins. (Bristol, England, John Wright, 1960.)
124. op. cit.
125. Ket, Y. et al. “Diabetes, Impaired Fasting Glucose and Development of Cognitive Impairment in Older Women.” Neurology. 2004;63:658–663.
126. Chatenoud, Liliane et al. “Refined-cereal Intake and Risk of Selected Cancers in Italy.” Am. J. Clin Nutr. Dec 1999;70:1107-1110.
127. Yoo, S. et al. “Comparison of Dietary Intakes Associated with Metabolic Syndrome Risk Factors in Young Adults: the Bogalusa Heart Study” Am J Clin Nutr. 2004 Oct;80(4):841-848.
128. Shaw, Gary M. et al. “Neural Tube Defects Associated with Maternal Periconceptional Dietary Intake of Simple Sugars and Glycemic Index.”
Am. J. Clin Nutr. Nov 2003;78:972-978.
129. Krilanovich, Nicholas J. “Fructose Misuse, the Obesity Epidemic, the Special Problems of the Child, and a Call to Action “ Am. J. Clin Nutr. Nov 2004;80:1446-1447.
130. .Jarnerot, G., “Consumption of Refined Sugar by Patients with Crohn's Disease, Ulcerative colitis, or Irritable Bowel Syndrome.” Scand J Gastroenterol. 1983 Nov;18(8):999-1002.
131. Allen, S. "Sugars and Fats: The Neurobiology of Preference." Am J Nutr.
2003;133:831S-834S.
132. De Stefani E, et al. “Sucrose as a Risk Factor for Cancer of the Colon and Rectum: a Case-control Study in Uruguay.” Int J Cancer. 1998 Jan 5;75(1):40-4.
133. Levi F, et al. “Dietary Factors and the Risk of Endometrial Cancer.” Cancer. 1993 Jun 1;71(11):3575-3581.
134. Mellemgaard A. et al. “Dietary Risk Factors for Renal Cell Carcinoma in Denmark.” Eur J Cancer. Apr 1996;32A(4):673-82.
135. Rogers AE, et al. “Nutritional and Dietary Influences on Liver Tumorigenesis in Mice and Rats.” Arch Toxicol Suppl. 1987;10:231-43. Review.
136. Sørensen L.B., et al. "Effect of Sucrose on Inflammatory Markers in Overweight Humans" Am J Clin Nutr. Aug 2005; 82(2)
137. Mohanty, Priya, et.al. "Glucose Challenge Stimulates Reactive Oxygen Species (ROS) Generation by Leucocytes," J Clin Endocrinol Metabol.. 2000, Aug:85(8) 2970-2973.
138. Arumugam V, et al. "A High-Glycemic Meal Pattern Elicited Increased Subjective Appetite Sensations in Overweight and Obese Women." Appetite. 2007; [Epub ahead of print].
139. Smith RN et al. "The Effect of a High-protein, Low Glycemic-load Diet Versus a Conventional, High Glycemic-load Diet on Biochemical Parameters Associated with Acne Vulgaris: A Randomized, Investigator-masked, Controlled Trial." J Am Acad Dermatol 2007;57:247-256.
140. Selva, D.M., et al. “Monosaccharide-Induced Lipogenesis Regulates the Human Hepatic Sex Hormone-Binding Globulin Gene.” J. Clin. Invest. 2007. doi:10.1172/JCI32249.
141. Fu M.L., et al. “Associatation Between Unhealthful Eating Patterns and Unfavorable Overall School Performance in Children.” J Am Diet Assoc. 2007;107(11): 1935-1942. 142. Krietsch, K., et al. “Prevalence, Presenting Symptoms, and Psychological Characteristics of Individuals Experiencing a Diet-related Mood-disturbance.” Behavior Therapy. 1988;19(4): 593-604.
142. Krietsch, K., et al. “Prevalence, Presenting Symptoms, and Psychological Characteristics of Individuals Experiencing a Diet-related Mood-disturbance.” Behavior Therapy. 1988;19(4): 593-604.
143. Drewnowski A. et al “Taste Preferences in Human Obesity: Environmental and Familial Factors. Am J Clin Nutr. 1991; 54: 635–641.
144. Berglund, M. et al. “Comparison of Monounsaturated Fat with Carbohydrates as a Replacement for Saturated Fat in Subjects with a High Metabolic Risk Profile: Studies in the Fasting and Postpr andial States.” Am. J. Clin Nut. Dec 1, 2007;86(6):1611 - 1620.
145. Ouyang X. et al. "Fructose Consumption as a Risk Factor for Non-alcoholic Fatty Liver Disease." J of Hepatol. 2008;48(6):993-999.
146. De Stefani E., et al. "Dietary patterns and risk of bladder cancer: a factor analysis in Uruguay." Cancer Causes Control, 2008; [Epub ahead of print].
Charles, let me turn the tables and ask you why you didn't do YOUR homework before you jumped into CureZone and with your very first post asked about issues that have been addressed and answered already? Sheesh!
Where to start . . .
OK, let's start with the human digestive system. When you ingest something it is digested in the stomach by digestive acids and broken down into constituent parts and then absorbed into the blood stream by individual molecules. There is no such thing as a maple syrup and sodium bicarbonate molecule and you can no more take the combination into the blood stream and to the cancer than you can take an oreo cookie there. Instead, the digestive system breaks down the combination into the sodium bicarbonate and the individual sugars and other components of maple syrup.
As for sodium bicarbonate, sure I think it can be useful when you can apply it directly to a tumor - such as a topical tumor, throat cancer or perhaps even cancers in the digestive tract possibly all the way to the rectum. Otherwise, you either have to inject it directly into the tumor or intravenously to have much chance of success.
If you were not a newcomer here you might know that in the years I have been here I have made several thousand posts either as myself (primarily in my own forum) or as my original ID Dquixote1217 in all the other forums. I don't make it a habit to attack anyone (except when provoked upon occasion) and I certainly don't attack someone who is an alternative cancer figure without doing my homework. When Simoncini burst upon the scene I thought that he and his baking soda therapy looked like hot stuff. The more I checked him out the more I found that a better description would be "steaming pile". I hate frauds when it comes to treating cancer, and that is what I largely consider Simoncini. Unlike what he has claimed, if he had a fraction of the success actually curing cancer as he has had promoting himself andhis book, he would be quite the wonder worker. But he hasn't, and as far as I can determine he has lied and deceived all along the way.
First of all, Simoncini could not have earned a fortune as an oncologist because Simoncini is NOT an oncologist, He is merely an MD, if that. He styles himself as an "MD who specializes in oncology". Even that might be debateable since it is noteworthy to see that nowhere on his own website or anywhere else that I can find does Simoncini list his educational credentials. Some speculate darkly about his education, given that Italy is known as a country where a "degree" can be had for the right amount of money.
Since I do not know about his educational background I will just say that I am suspicious until he is more forthcoming, and, regardless of his background, I still have little regards for the man. I know people who made the trip to Italy to see the famous Simoncini and came away having less than stellar regards for him and complained about what they most noticed was his incompetence and lack of professionalism. Here is the report one of them made on my Yahoo health group back when people were first oohing and aahing about Simoncini:
How can I say this without sounding like a naysayer? Last summer my wife and I were in Rome where we lived for four months. It was during that time that I first read about this doctor and so I made an appointment and went to see him. What I had read sounded plausible. My condition is chronic lymphocytic leukemia known as CLL.
When I went in to see Dr. Simoncini, I brought with me reports of several blood tests. He looked these over and then took my pulse. He may have looked at my tongue as well. That was the extent of the examination. He indicated that he didn't feel that I had CLL at all. I was really quite astounded to hear this because my diagnosis was confirmed by the National Institute of Health. I'm not saying that NIH never makes mistakes; however, the analysis of the bone marrow tests and the blood work and the lymph node analyses were used to come up with the diagnosis.
Dr. Simoncini may be onto something here, though, when it comes to cancers that produce solid tumors such as breast, throat, and so on. As an oncologist, however, I expected a little more than a flip response like that.
Caveat emptor.
Allen
Another person told me that Simoncini seemed more interested in talking about himself than he did in diagnosing or helping them.
Simoncini fled Italy after being charged with manslaughter in the deaths of two patients, and went to the Netherlands where he infamously treated several patients at a cancer clinic with the result that no one was cured and one patient died after Simoncini repeatedly injected sodium bicarbonate into her breast. The diagnosis was that she died from acute alkalinosis. Evidently, Simoncini beat a hastry retreat once again and at last report her death was also being investigated with an eye towards charges being filed. I might give him the benefit of the doubt and say he was just another brave pioneer who was persecuted by the mainstream medical establishment but . . .
It turns out that Simoncini used fake testimonials to promote his so-called successes. One woman who claims to have been cured turned out to have never had cancer to begin with. Another testifier for Simoncini was later identified as his mistress. And a third, who was perhaps Simoncini's top poster girl, later complained bitterly that she had been deceived by Simoncini into believing her cancer was cured when it fact it was progressing and by the time she found out it was too late for any alternative that might have saved her life. She died a few months ago and left two small children without their mother.
So, don't invite me and Simoncini to the same dinner party - or expect me to have anything positive to say about him.
Anyway, I hope I have answered your question. If not, I am sorry, but I am thoroughly tired of cussing and discussing Simoncini.
Welcome to CureZone and all the best to you,
Tony