More specific info:
re: my post earlier May 12 about the mitochondria being blocked from full ATP energy output due to toxins or lack of specific active adenosyl B12, here's a short article on that. it looks like what you describe for symptoms: http://vitaminb12deficiency.net.au/VB12Energy.htm
Deficiency in adenosylcobalamin leads to the accumulation of methylmalonic acid, which disupts normal glucose and gluatmic acid metabolism in the cell due to its inhibitory activity on the Krebs cycle and by inhibition of ATP synthase. Continued deficiency of adenosylcobalamin, with resultant reduction in energy output can lead to anorexia, lacrimation, alopecia, and eventual emaciation. In addition there is a build up of lesions in the liver and the development of optic neuropathies