Good link about brain/colon link, thanks for that...I need to read it again with more time, as there is quite a lot of info there. I will need to get more info on the internal zapper, although I havent yet seen how zapping works for me. I dont have a zapper (yet), neither an ozonatos (BTW, anyone has a link to where to get reliable ozonatos on the net??).

About 2.5 years ago I had the sensation of "things" going up & down my spinal cord, like if it was being used as a motorway. I had very heavey headaches and pain in the nuchae area. At that time I had -and still have- very high levels of anti-thyroid antibodies. So I "rationalized" this sensation as if maybe the antibodies where having a ride on the spinal cord (didn't mentioned it to anyone as I felt it will be misinterpreted, only mentioned it in the support forum I used). Last year I had an brain MRI due to pain and numbness in my face, and it showed a thickening of the meninge on the retrocerebellar midline; initialy I was told it could be a meningioma, but with contrast on it did not show active, so it was 1) ignored 2) put down to fat(?) 3)put down to a thickening of the dura matter (by 3 different consultants). The one that seemed more "rational" to me was the 3rd one.
Now I think I should include the parasites connection to these explanations.
It will be interesting to see whether my antibodies will be affected by the protocols I am following now.
FireEarth...

Hi FEWA, I found a small clip from an article which I think gives a basic explanation of how this works. The innate immune system is complex, but it is safe to say, that when cells of the mucosal lining in the intestine encounter something with antibodies on it, it will secrete mucins with the intent of surrounding the foreign object with a slippery coat to contain and expel it.pmc logo image
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Note: Performing your original search, antibodies intestinal mucus, in PubMed Central will retrieve 1611 citations.
Journal List > Immunology > v.70(1); May 1990
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PubMed articles by:
Carlisle, M.
McGregor, D.
Appleton, J.


Immunology. 1990 May; 70(1): 126–132.

PMCID: PMC1384093
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The role of mucus in antibody-mediated rapid expulsion of Trichinella spiralis in suckling rats.
M S Carlisle, D D McGregor, and J A Appleton
James A. Baker Institute for Animal Health, Department of Microbiology, Immunology and Parasitology, New York State College of Veterinary Medicine, Cornell University, Ithaca 14853.
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Abstract
Rat pups suckling dams parasitized by Trichinella spiralis express rapid expulsion, a protective response that is associated with the entrapment of infectious muscle larvae in intestinal mucus. Immunofluorescent studies revealed that antibodies were bound to the surfaces of the entrapped larvae. Mucus binding and rapid expulsion occurred in normal pups dosed with larvae coated with antibodies prepared from infected rat serum. Subsequent experiments revealed that entrapped larvae escaped from mucus after 2 hr in vitro incubation in saline. Escape correlated with the loss of the surface-bound antibodies, suggesting that mucus entrapment was reversible and dependent on antibody coating. Finally, when protective antibodies were injected 1, 2 or 6 hr after larvae were administered to pups, the parasites were forced to leave their epithelial niche and became enveloped in mucus. The above findings suggest that mucus trapping of T. spiralis larvae is dependent upon the coating of larvae by antibody, but that trapping is reversible, and is not in itself the pivotal event in rapid expulsion. The primary mechanism of rapid expulsion appears to be antibody-mediated inhibition of processes required for the parasite to maintain itself in the epithelium.
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Selected References
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