Mycotoxins – An Overlooked Problem

http://fungifinder.com/toxicmold.htm

This is an excerpt from the Finnish Journal of Environmental Medicine - Establishing effective protocols for systemic fungal disease; especially in North American patients

Systemic Fungal Disease - Mycotoxicosis:

Ill patients approached us from an evacuated apartment building with several million IAQ counts of toxigenic fungi.  These were the highest counts we had ever seen.

In this study, most of whose host defense mechanisms were compromised from toxigenic fungal exposure ranged from minor to fatal and often are caused by organisms that normally reside on or inside body surfaces. In the hospital setting, they frequently result from colonization by antibiotic-resistant organisms (opportunistic fungi) and unskilled physicians who often misdiagnose, disregard, and prescribe drugs that can be potentially detrimental to the immunocompromised  hypersensitized  patient who often are unknowlingly poisoned by the to T-2 mycotoxins they have been exposed to in the sick building they have been exposed to.

The American medical industry, who is generally trained in finding a “quick fix” to the immediate symptoms with often ineffective pharmaceuticals, rather than finding a permanent corrective action plan.  This generates a great disservice to the general public, who is basically trusting and ignorant to doctors upon their initial consultation, until they discover that these placebo treatments are ineffective and the patient is making no progress; sometimes too late in regards to finding aggressive and alternative treatment options for these patients who are at times, gravely ill.

Host defense mechanisms--physiologic, anatomic, or immunologic--may be altered or breached by disease or trauma or by procedures or agents used for diagnosis or therapy. Infections in this setting, often called opportunistic infections, occur if antimicrobial therapy alters the normal relationship between host and microbe or if host defense mechanisms have been altered by age, burns, neoplasms, metabolic disorders, irradiation, foreign bodies, immunosuppressive or cyto toxic drugs, corticosteroids, or diagnostic or therapeutic instrumentation.  This ideology has changed over the past five years as patients with normal health histories have incurred systemic fungal infections with exposure to lethal fungi such as stachybotrys and chaetomium causing damage to the myelin sheath.  These otherwise normal patients became gravely ill and autoimmune disease was diagnosed within one year to eighteen months on average in 68% of the patients who were exposed to these to fungi types.  In the other patients, systemic fungal disease was diagnosed in 32% with minor symptoms, including reactive airway disease, asthma, GERD, joint/muscle pain, memory problems, balance, vision, and hypersensitivity.  Colonization occurred in 46% of these cases and aggressive antifungal protocol had to be followed.  All patients followed a rigorous vitamin, enzyme, diet therapy that was highly effective for 97% of all cases when administered in a timely manner.   

The underlying alteration predisposes the patient to infections from endogenous micro flora that is nonpathogenic or from ordinarily harmless, saprophytic organisms acquired by contact with other patients, hospital personnel, or equipment. These organisms may be bacteria, fungi, viruses, or other parasites; the precise character of the host's altered defenses determines which organisms are likely to be involved. These organisms are often resistant to multiple antibiotics, as they contain fungi.

Drug Therapy and Impaired Host Defense Mechanisms

Antibiotics alter the normal micro flora of the skin, mucous membranes, and GI tract and may result in colonization by new organisms. Colonization is harmless unless followed by super infection, which refers to invasion by indigenous or environmental organisms resistant to the administered antibiotic. Factors predisposing to super infection include extremes of age, debilitating diseases, and prolonged treatment with antibiotics, especially broad-spectrum ones. Super infections usually appear on the 4th or 5th day of therapy and may convert a benign, self-limited disease into a serious, prolonged, or even fatal one. The diagnosis of super infection by a normally commensal organism is certain only when the organism is recovered from blood, CSF, or body cavity fluid. 

Corticosteroids, often mistakenly prescribed by unskilled or uninformed physicians looking for a “quick fix,” alter many aspects of host defenses; one of the most important is inhibition of the movement of neutrophils, monocytes, and lymphocytes into the inflammatory exudate. Corticosteroids may reactivate quiescent pulmonary TB, histoplasmosis, coccidioidomycosis, and blastomycosis. Patients receiving corticosteroid treatment (especially in high dose) for RA, ulcerative colitis, asthma, sarcoidosis, SLE, pemphigus, or Cushing's syndrome have increased susceptibility to infection from usual and unusual bacteria and tend to develop infections with chaetomium and stachybotrys, as previously mentioned. 

Aggressive treatment is necessary to end myco toxic osis. There is not a drug available to cure the problem but many help. Antifungals are much more beneficial than cholestyramine. If one wants to create anoxynase in the body, there are far more healthier and economical ways to do it than administering harsh chemicals in the body. There is currently a vitamin/enzyme/mineral/electrolyte therapy that is very helpful but has not been made available for clinical trial yet.  It will soon be available.  There are many treatment options available on this website that is approved and very beneficial, and at no charge from us. More updates will be published soon.