Chitin - Chitinase - Lectins - anyone researched?

Can anyone help me make sense of this?

Components of fungal cell walls:

Chitin-N-acetylglucosamine
Glucans
Ergosterol

Chitinase being the enzyme to break-down chitin.

Seems chitinase could help with candida fungus/hyphae/pseudohyphae...the fungal form, not yeast form.

So i'm following this path of research...and get to Lectins.

Lectins found mainly in grains, beans, seeds, potatoes, tomatoes.

Dietary lectins generally cause inflammation, hormone disruptions, metabolic disruptions, Bowel problems, interfere with protein digestion, increases gut permeability etc.

Lectins bind to chitin-N-acetylglucosamine. (for a moment i was wondering if consuming lectins would HELP bind candida!)

Then i stumble upon a pubmed study - http://www.biomedcentral.com/1471-2180/8/229


"Mannose binding lectin (MBL)[1], a plasma protein of hepatic origin that belongs to the family of calcium-dependent collagenous lectins (collectin), is an important protein of the innate immune system [1-6]. This carbohydrate-binding protein binds mannose and N-acetylglucosamine (GlcNAc) sugars and their derivates present on the surface of a wide range of clinically relevant microbial species and has the ability to distinguish self from nonself [4,5,7,8]."

"Binding of MBL to different pathogens

A striking difference in binding patterns of MBL to the different microorganisms was found (Figure 1). Binding of MBL to C. albicans, C. parapsilosis and in a lesser extend to acapsular C. neoformans was found. Almost no binding of MBL was observed to S. aureus, E. coli and encapsulated C. neoformans. No binding was observed in the absence of Ca2+ (data not shown) and when purified MBL was not added. "

"MBL is an important opsonin for phagocytosis of Candida species and acapsular C. neoformans. It lacks the function as an opsonin for phagocytosis of encapsulated C. neoformans.

These data suggest the importance of MBL in the first-line defence against Candida species and acapsular C. neoformans. "

So if a person is deficient in MBL due to gene defect, sluggish liver, immunosuppresion etc - how would we introduce more Mannose binding Lectin? Or improve it's production as it's not really available widely to take as a supplement - as it needs storing at -70 and administered carefully!
Dietary and hepatic lectin seem to differ...and as i've stated above - dietary lectin seems to induce bowel inflammation, colitis, etc, so does not do the job of MBL.

I'm considering that candida could possibly CAUSE a MBL deficiency and sluggish liver.

Is this a valid candida -busting route for the hyphae fungal form of the species?

Anyone else gone down this route already? Brainfog is inhibiting alot of assimilation of info at the moment, i felt i was onto something, then i ended up in a pit of confusion!

Experience and success of chitinase would be welcomed too.
Thanks in advance...:-)