whole text by JulieAKA87980 ..... Parasites Debate Forum
Date: 10/30/2008 9:44:53 AM ( 16 y ago)
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URL: https://www.curezone.org/forums/fm.asp?i=1288425
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This is the contents of toxic worm.com, I got on this morning, and saved the whole thing:
(I have a feeling Dr. Harvey may well have got his idea from my website. What do you think?.........)
The following is an exert from the Washington Post.
"William Harvey, 70, who serves as chairman of the MRF board, has taken those theories one step farther. He says he became interested in Morgellons research after successfully battling chronic fatigue syndrome and made it his mission to find cures for such unexplained illnesses.
He wouldn't be specific, explaining that he first wants the results of his research to appear in a top-notch, peer-reviewed journal such as the Lancet. "This may be the story of the century," he says. A semi-retired doctor in Colorado Springs who spent most of his career working in space medicine for the Johnson Space Center, Harvey says he may have found not only why Morgellons patients would both scratch and act strange, but also what could be the "genesis of probably most chronic human illnesses," such as autism, obesity, chronic fatigue and bipolar disorder.
It all boils down to this: mutant worms.
Harvey hypothesizes that a type of nematode, a wormlike parasite that lives in the soil as well as in the guts or lungs of about half the animals on the planet, mutated somewhere in the 1970s in Southeast Asia and jumped from animals to humans. The parasite is easily spread through the fecal-oral route if someone, for example, is out working in the garden, fails to wash his or her hands thoroughly and then eats an orange. Or it gets into the lungs by inhaling sputum or by kissing. The worm then takes up residence in the colon, Harvey theorizes, and the body's immune system holds it in check.
But when the immune system falters, the worms swarm in the body. That's what happens, Harvey hypothesizes, after a human is infected with a strain of bacteria first reported in 1986, Chlamydophila pneumonia. These bacteria like to live in immune cells, Harvey says, and they feast on those cells' energy. With the host's immune system compromised, the mutant nematodes begin reproducing exponentially, Harvey suspects. They burrow a hole in the wall of the colon, then usually travel at night through the bloodstream or the lymphatic system or crawl in hordes between the layers of the skin, like other species of nematodes are known to do, to the parts of the body with the most blood flow: the face, head and nose. There, a cranial nerve leads right into the brain. A pileup of worms could jam blood and oxygen flow to the brain, Harvey says. "That may explain the psychological symptoms," including the hallucinations, he says......."
..."The fibers, according to Harvey's theory, are really the hard shells, which he calls cuticles, that these worms shed at five stages as they grow from egg to larvae to adult. The red fibers are the males, he says. Blue fibers are female. "Using a 2,000-power microscope, you can see inside them," he says. "They look like little stovepipes to me. I can tell the blue ones are female because there's a kink in the middle for the sexual organs and some kind of pouch. And we have pictures of them laying thousands of eggs."
"If you write this theory, it's probably going to sound like someone's come from the mental institution," Harvey says. "But the fact is that this is a real disease, and it appears to be growing."
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A SILENT PANDEMIC
The changing face of humanity
Synopsis
A pandemic of chronic infection by a newly discovered species of a hormone-secreting nematode (roundworm), "Cryptostrongylus Pulmoni", which colonises several regions of the body - and yet whose primary site of colonisation is the lower spine or lumbar lymph node - has been persisting undetected by doctors, scientists and health authorities.
The most likely reason it has persisted undetected is because the parasite manages to suppress an immune response by the host. Much like invisble ink, the exact site of chronic infection only becomes apparent when an agent has been applied.
As a chronic spinal infection caused by this toxic parasite passes through a community - indeed a nation – it's broad range of primary and secondary affects are being systematically misinterpreted as a series of unrelated ailments.
The notion of this kind of professional misinterpretation surrounding parasitosis can be supported by a precedence regarding outbreaks of another species of nematode, “Trichinella Spiralis”.
It has been documented that symptoms directly relating to the disease caused by the trichinella worm, trichinosis, have, on occasions, been misdiagnosed by doctors as incorrectly relating to as many as fifty individual, unrelated diseases.
As an example, there have been reported instances where patients suffering from trichinosis have been misdiagnosed as suffering from non-infective mental disorders, since the trichinella worm has been known to sometimes migrate through the cerebrospinal fluid, causing hallucinations in its host.
It's therefore possible the same diagnostic phenomenon – the perception of the ‘one as many' - could also be occurring in association with a pandemic of chronic infection by the largely unknown toxic nematode, Cryptostrongylus Pulmoni.
Chronic infection, extending to the heart, the genitourinary tract, the kidneys, adrenal glands, and, primarily, the lower spine - infection via the rectum
Female infection via the rectum
Infection spreading to the spinal column and genitourinary region Male infection via the rectum
Colonies or cysts occurring adjacent to the prostate gland and close to the anus may be misdiagnosed by doctors as "haemorrhoids"
Colonization by a tissue-boring parasite in the region of the anal passage, rectum and bowel, through its close proximity, may gain access to the region of the genitourinary tracts in both males and females (prostate, seminal vesicle, vagina, uterus, bladder). Although colonization may be chronic, symptoms may manifest episodically as 'burning' micturition (urination), and occasional pain or cramps in the region of the prostate, testicles (epididymitis) or vagina.
Colonization by a tissue-boring parasite in the rectum, in close proximity to the spinal column (i.e. lumbar lymph nodes), could manifest as either: episodes of a sudden, sharp 'grabbing' pain (particularly during the initial 'invasive' stage); chronic pain in the lower back; occasional severe leg muscle cramps; a chronic arthritic condition; or general rapid deterioration of the spinal column marked by the narrowing of a disc space.
Chronic discitis or disc space infection may result in infection of the cerebrospinal fluid (CSF) - complications known to be occurring in association with other nematode infections, like trichinosis.
It's probable any clinical evidence indicating widespread rapid deterioration of the spine would also be interpreted by doctors as muscular or skeletal and " a combination of both genes and environment", once again implying a non-infectious nature of the condition.
This interpretation can be further strengthened when a chronic infection in the region of the lower spine is 'stirred up' or aggravated due to specific environmental stresses (like bending over), leading the sufferer to believe the back problem is non-infective.
Other symptoms of colonization in the rectum may occur as profuse bleeding from the anus. A colony or 'cyst' of nematodes in the rectum or anal canal, if observable at all, would most likely be diagnosed by doctors as simply "haemorrhoids".
An eruption of boils and blisters revealing the alleged primary site of colonisation - and the alleged site of reproduction induced via the application of a low voltage electric current
The photo above demonstrates the alleged primary site of colonisation of this parasite - the 'central station' of the infection - most likely the lumbar lymph node.
This massive eruption of boils and blisters in the region of the coccyx and lower spine was induced by simply passing a low voltage current of electricity through a region of chronic pain or stiffness in the lower back.
My suspicion was such that when the main site of reproduction of this infection - the 'epicentre' - was being attacked by either the immune system or medication, it sets up an inflammatory response that somehow serves to protect it.
Since electricity is a force capable of penetrating beyond any physical or chemical barriers (providing there is sufficient conductivity), I experimented by applying a 6 volt (or a 9.6 volt) 0.75 amp current to find out if it may indeed penetrate this inflammation, and thereby render an accompanying medication more effective.
Basically I wanted to see if it would induce a migration of a suspected parasite.
It did. (...and how!)
The 'craters' you see in this photo are blisters and boils that had been cleaned out with hydrogen peroxide. As you can see, they extended deep towards the spinal column
If a biopsy of flesh were to be conducted (cut deep towards the spinal column) my prediction is that here will lie the answers to many previously unanswered questions.
This is a very serious chronic spinal infection that, unbelievably, has been occurring as a global pandemic and most likely capable of influencing to a marked degree the collective psyche of an entire species. (re: low grade to acute encephalitis manifesting as a psychotic or neurotic 'rabies-like' mental state characterised by paranoia (it's paranoia that makes a rabid animal aggressive); aggression; obsession; and depression.
The region of infection of the lower spine appears to also extend to the region of the kidneys and adrenal glands, however, which may account for a suspected hormonal interference and associated fluctuations in body weight - usually weight gain. Such an interference in adrenal hormone production may instead account, therefore, for the symptom of episodic aggression.
Parasitosis and the predatory food chain
The perspective of medical researchers or psychologists rarely takes into account factors related to ecological forces. The living, working and educational environments of these professionals is generally culturally urbanized. Because of this underestimated yet arguably significant factor, there is a tendency for health problems, such as obesity, psychoses, depression, or heart disease, to be interpreted as relating to societal (or congenital), rather than ecological circumstances.
One species of roundworm's only means of transmission from one host to the next is when one host is eaten by the next. The trichinella worm produces cysts or “nurse cells” embedded in the muscle fibre of the host, and when a predator eats those cysts, the worm is then released into the stomach of its next host.
It's therefore suspected by scientists that the trichinella worm may actually create health conditions in its host that leave it more vulnerable to attack by a predator, thus ensuring its own parasitic survival. These health conditions that create this kind of vulnerability may relate to both physical as well as psychological impairment. Since the trichinella worm sometimes migrates through the cerebrospinal fluid, it can cause hallucinations in its host. A host mentally disorientated in such a way is rendered much more vulnerable to attack by a predator. In a similar manner, predation would presumably play a key role in the transmission of many other parasitic diseases.
One can be sure that a rabbit that develops the chronic fatigue syndrome is the rabbit that will inevitably be caught by the fox. If a parasite, such as C. Pulmoni (or "the Morgellons Worm"), causes chronic fatigue, then the physical vulnerability it creates in its host may also be playing a key role in the transmission of the nematode down through the predatory food chain.
Nature is quite explicit and deliberate in its evolved designs: any animals in the wild that are either psychotic or chronically fatigued, without exception, share only one fate: They all get eaten.
If the toxic, hormone-secreting C. Pulmoni, in some cases, may cause periodic manifestations of psychosis or other aberrant behaviourial patterns in some individuals (via a chronic infection of the lower spine - its primary site of colonisation, as well as its primary systemic influence), then those hosts may also be socially alienated from the protection of the herd, isolated, and once again rendered much more vulnerable to attack by a predator.
The broader the range of potential reservoir hosts, the greater the capability for it to proliferate human populations. If for various technical or subjective reasons a species of nematode is not being identified in humans, then it would probably also be remaining undetected in animal hosts for the same reasons.
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Blood tests generally appear normal: eosinophilia is usually absent. Ige may be normal. ESR is only sometimes moderately increased.
In fact, there would be no blood test that could identify chronic infection by this particular species of parasite.
* The only effective and conclusive method would be physical examination of sputum, blood, lymphatic fluid (in particular, the lumbar lymph nodes), or cerebrospinal fluid.
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Cryptostrongylus Pulmoni, the “hidden lungworm”
Diagram 1
Micrograph 1
Micrograph 2
This is how Micrograph 2 may appear with the red colour intact.
An approximation of Dr. Klapow's portrayal of the shape of the male C. Pulmoni. For a more detailed and accurate diagram, refer to Dr. Klapow's own diagram on his website (web address listed below)
This red object, identified in a local blood sample (from the Armidale region) is consistent in shape, size and colour to the male C. Pulmoni. The posterior is in accordance with the shape and proportional size of the copulatory bursa of the male C. Pulmoni, as documented by Dr. Klapow.
This minute bulbous object with a fine whip-tail was identified in a local blood sample. The significance of it lies in the tiny remnants of coloured pigment (re: arrow). There is no doubt this object was once a bright red/pink colour before the pigment was washed out. It appears to be an organism bearing a flagellum of some sort, implying locomotion. Really I've only included this object because it's tendency towards significance outweighs it's tendency towards irrelevance
Cryptostrongylus Pulmoni
Cryptostrongylus Pulmoni is a nematode, or roundworm, that is very small and not visible to the naked eye. The bright red male measures only 200 to 350 microns in length, while the female is a little less than one millimeter long. Its name means “hidden lungworm” because it has been recovered from upper respiratory sputum coughed up from the lung of chronic fatigue syndrome (CFS) patients.
The American invertebrate biologist, Dr. Lawrence Klapow, discovered this new species of roundworm several years ago and conducted preliminary research into it in conjunction with Harvard University. He claims this worm is colonizing a projected 63% of Chronic-Fatigue-Syndrome patients (given test sensitivity). The anatomy of the sex organs of this worm implies it belongs to a family of nematode, trichostrongylids. It's suspected the parasite may have originated from the jungles of South East Asia. Trichostrongylids are known to secrete various hormones and cytokines. The two main hormones or peptides secreted by trichostrongylids are “hippocampal cholinergic neurostimulatory peptide” (H.C.N.P.), and “vasoactive intestinal polypeptide” (V.I.P.).
• H.C.N.P., as the name suggests, is a neuropeptide affecting the hippocampus, the region of the brain associated with memory. The affects of this hormone appear to be, once again, consistent with symptoms related to memory loss which have been commonly experienced, in varying degrees, upon a widespread basis about the New England region (and most likely other regions of Australia). An extreme form of memory loss is Alzheimer's disease. Alzheimer's disease also appears to have been quite frequently diagnosed over the last seven or eight years around the New England region (Australia).
• V.I.P. is a hormone which, as the name suggests, affects blood pressure - particularly blood flow to the brain. The discreet production of this peptide directly deposited into the CSF by a trichostrongylid appears, once again consistent with the simultaneous and widespread development of hypertension (or conversely, hypotension) and other cardiovascular disorders (including stroke) among many locals in the New England region.
• V.I.P. is also known to influence the hypothalamus, and therefore hormone production of the HPA-axis, such as pituitary function (and the production of the hormone, CRH (ACTH), or ‘cortisol regulating hormone')
The toxic affects of these hormones and cytokines would be much more powerful if they were being secreted directly into the cerebrospinal fluid, as may be the case with a chronic spinal infection.
To find Dr. Klapow's website: click here (for further information about Cryptostrongylus Pulmoni, see the transcript of a radio interview with Dr. Klapow on a related website)
An apology
Dr. Klapow would most likely personally distance himself from my own independent claims of a widespread pandemic of this toxic parasite, and the extent of the broad range of symptoms I am alleging it can cause. He is a professional scientist that adheres to strict scientific protocol, whereas I'm an uneducated farmer who just happens to believe he is noticing something important that's being seriously underestimated. As such, Dr. Klapow would probably view these scientifically unproven claims and actions to draw public attention to the regional presence of this toxic nematode by compiling this website as possibly inflicting damage upon the general professional credibility of his own research. This has obviously not been intended.
On the other hand…
With respect to just a few technical aspects, I allege that Dr. Klapow's own research has arguably been flawed. Whilst documenting the female C. Pulmoni and claiming the specimens he recovered were “isolate decayed”, he evidently had not made any adjustments or speculations as to how a female nematode in a ‘fresh' form might appear. When Dr. Klapow was aware that the bright red colour of the male C. Pulmoni, under certain circumstances, can be washed out leaving it totally clear, he didn't consider the same phenomenon of colour loss could also be occurring with his partially decomposed female specimens. As mentioned later, I believe Dr. Klapow got the portrayal of the female C. Pulmoni wrong: I think the female, like the bright red male, also evolved a bright colour – but instead, she possesses a deep, sapphire-blue pigment (re: micrograph 3 – see later).
The structure of the mouth parts of the female is also significant. Hypothetically, mouth appendages of a rigid nature would most likely always change into a soft and floppy form as they deteriorate through the digestive affects of the sputum. If the mouth parts that Dr. Klapow has labeled the “six prehensile lips” were, as I have later described, rigid (and not soft and floppy as Dr. Klapow has documented them), then this may present a stronger likelihood of the nematode as a 'tissue borer', and hold ramifications as to the extend the parasite can disseminate to other regions of the body (like the base of the spine) - regions that Dr. Klapow failed to consider.
Sorry this is without the photos, but at least I have the whole text.
Julie
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