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Re: h.pylori
 
jessesmom1987 Views: 2,484
Published: 17 y
 
This is a reply to # 1,175,056

Re: h.pylori


We'll see what Charlie has to say, but I have been thinking about it recently too, and would like to know what Charlie has to say about taking Betaine.



http://www.en.wikipedia.org/wiki/Helicobacter_pylori



Just a few words from the start of the description-
Helicobacter pylori (pronounced /ˌhɛlɪkəˈbæktɚ paɪˈlɔəraɪ/) is a gram-negative, microaerophilic bacterium that infects various areas of the stomach and duodenum. Many cases of peptic ulcers, gastritis, duodenitis, and cancers are caused by H. pylori infections. However, many who are infected do not show any symptoms of disease. H. pylori's helical shape (from which the genus name is derived) is thought to have evolved to penetrate and favor its motility in the mucus gel layer



They treat it with a course of anti-biotics after testing positive...my dad went through it.

However:


In recent years, Helicobacter pylori (H. pylori) has been identified by researchers as the cause of the majority of gastrointestinal ulcers. H. pylori is a bacterium that lives in the stomach and duodenum. It has a unique way of adapting to the usually harsh environment of the stomach.



Incidence; Causes & Development


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H. pylori is very common, infecting more than a billion people worldwide. It is estimated that half of the United States population older than age 60 has been infected with H. pylori. Infection usually persists for many years, leading to ulcer disease in 10 % to 15% of those infected. H. pylori is found in more than 80% of patients with gastric and duodenal ulcers.

Gastric juice is composed of digestive enzymes and concentrated hydrochloric acid, which can readily digest food or kill microorganisms. Low levels of stomach acid increase the chance an organism's survival. It used to be thought that the stomach contained no bacteria and was actually sterile.

The stomach is protected from its own gastric juice by a thick layer of mucus that covers the stomach lining. H. pylori takes advantage of this protection by living in the mucous lining.

Once H. pylori is safe in the mucus, it is able to fight the stomach acid that does reach it with an enzyme it possesses called urease. Urease converts urea, of which there is an abundant supply in the stomach (from saliva and gastric juices), into bicarbonate and ammonia, which are strong bases. This creates a cloud of acid-neutralizing chemicals around the H. pylori, protecting it from the acid in the stomach. The breath test method of diagnosis relies on this reaction being present.

Contributing to the protection of H. pylori is the fact that the body's natural defenses cannot reach the bacterium in the mucous lining of the stomach. The immune system will respond to an H. pylori infection by sending white cells, killer T-cells, and other infection-fighting agents. However, these potential H. pylori eradicators cannot reach the infection, because they cannot easily get through stomach lining. They do not, however, go away - the immune response just grows and grows. White cells die and spill their destructive compounds (superoxide radicals) on stomach lining cells. Extra nutrients are sent to reinforce the white cells, and the H. pylori can feed on this. Within a few days, gastritis and perhaps eventually a peptic ulcer results. It may not be H. pylori itself which causes peptic ulcer, but the body's response (inflammation of the stomach lining).

H. pylori is believed to be transmitted orally. Many researchers believe that H. pylori is transmitted orally by means of fecal matter through the ingestion of waste-tainted food or water. In addition, it is possible that H. pylori could be transmitted from the stomach to the mouth through gastro-esophageal reflux or belching, common symptoms of gastritis. The bacterium could then be transmitted through oral contact.



 

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