MDs have evidence candida growth in the gut.!

Chronic intestinal candidiasis as a possible etiological factor in the chronic fatigue syndrome.
Cater RE 2nd.
Abstract
The chronic candidiasis syndrome, also known as the Candida-related complex, putatively caused by the overgrowth of Candida albicans in the gastrointestinal tract and secondarily in the genital organs, is briefly described. Patients with this disorder have many of the same symptoms as those with the chronic fatigue syndrome, except for the recurrent flu-like symptoms of the latter disorder. The positive response of a large number of patients with the chronic fatigue syndrome (CFS) to an oral antifungal agent and a diet for intestinal candidiasis has been described by another clinician. There is evidence that Candida albicans infection of the mucous membranes depresses T cell and natural killer (NK) cell function. Similar abnormalities of immune function are found in the CFS. The function of cytotoxic T cells, T helper cells, and NK cells is important in preventing reactivation of infections from Epstein-Barr virus, cytomegalovirus, and other herpesviruses. Reactivation of one or more of these viruses could lead to the expression of the flu-like symptoms in the CFS. Yet the immune dysfunction found in this disorder has been considered the primary underlying causal factor. It is proposed that chronic intestinal candidiasis may be an agent which leads to immune Depression in many Chronic-Fatigue-Syndrome patients and therefore that it could be a causal factor in CFS.

Fungal infections of the small and large intestine.
R J Prescott, M Harris, and S S Banerjee
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Abstract

AIMS: To study the pathological features of fungal infections affecting the lower intestinal tract (duodenum, small and large bowels). METHODS: Between mid-1981 and mid-1991, 14 cases of deep mycotic infections affecting the lower intestinal tract were found among 890 consecutive necropsies on patients with malignant disease treated in a regional cancer centre (incidence 1.6%). These 14 cases accounted for 54% of all gastrointestinal fungal infection detected. The relevant clinical, necropsy, histological and microbiological data were reviewed. RESULTS: Candida spp and Aspergillus spp accounted for all infections. The macroscopic appearances included ulcers of varying configuration, mucosal flecks, sloughed mucous membranes, polypoid masses and segmental lesions. Either organism could produce this range of lesions, but Candida tended to have a mucosal location and Aspergillus was associated with transmural invasion. Combined infections showed Candida in the surface mucosa and Aspergillus hyphae in submucosal vessels with spread into the bowel wall in a radiating pattern. During the final illness, gastrointestinal symptoms and signs were often slight and microbiological investigations were unhelpful. CONCLUSIONS: Variable gross appearances are relevant for endoscopists, particularly lesions which resemble pseudomembranous colitis. Endoscopic biopsy specimens may have a role in antemortem diagnosis. Failure to diagnose these infections during life emphasises the importance of necropsy in the clinicopathological audit of deaths in this group of patients.

SUCCESSFUL TREATMENT OF INTESTINAL MONILIASIS WITH FATTY ACID-RESIN COMPLEX
IRENE NEUHAUSER, M.D.
AMA Arch Intern Med. 1954;93(1):53-60. doi:10.1001/archinte.1954.00240250063005. Text Size: A A A
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ABSTRACT
ABSTRACT | REFERENCES
INTESTINAL infections due to Candida albicans have recently become a subject of much interest.1 The appearance of large numbers of this organism in the feces of patients receiving certain Antibiotics was noted soon after the introduction of these drugs into clinical medicine.2 The cause of this change in the composition of the intestinal flora lies in the profound effect which several Antibiotics have on the normal intestinal bacteria and the almost complete resistance which C. albicans and certain bacteria show towards the action of these drugs. The great decrease in number of the normal intestinal bacteria brought about by the Antibiotic drugs may play a role in the rapid increase in the number of C. albicans organisms, since the intestinal wall areas, previously occupied by these bacteria, may now become available as a source of nutrilites to organisms resistant to the drugs.3 Indeed, there has been a