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Re: the permeability of the blood-brain barrier & MSG *edit* by wombat ..... Iodine Supplementation Support by VWT Team

Date:   7/7/2007 9:34:55 AM ( 17 y ago)
Hits:   5,563
URL:   https://www.curezone.org/forums/fm.asp?i=914219

From "Excitotoxins, the Taste that Kills" by Russel Blaylock, MD.:

"It may well be that as these Parkinson's patients age and develop small strokes, hypertension or other diseases of the brain, their blood-brain barrier becomes more porous, allowing excitotoxins in the blood to enter. By ingesting diets high in MSG, hydrolyzed vegetable protein, Aspartame and other excitotoxins, these people would be exposing their brains to damaging levels of this class of toxin. Eventually other neurons would be damaged, possibly leading to symptoms of Alzheimer's type dementia and ALS.

It is also possible that glutamate and aspartate can enter the brain WITHOUT A BREAKDOWN IN THE BLOOD-BRAIN BARRIER. We know that certain parts of the adult brain have no, or incompetent barriers. These areas are near the ventricular system(circumventricular organs) and can potentially act as points through which glutamate and aspartate in the plasma could slowly seep into the surrounding brain, thereby BYPASSING THE BLOOD-BRAIN BARRIER. This would explain the delay in brain accumulation of radioactively labeled Mono-Sodium-Glutamat (Natrium Glutamat) following it's injection in animals."

*edit* more...

"There is another bit of evidence that points to excitotoxins as a major contributor to the causation of Parkinsonism. A recent analysis of Parkinson's patients disclosed that the loss of neurons is a linearly progressive pathological process. This means that the number of neurons lost is the same every year. Such evidence is not compatible with a disease that suddenly accelerates or decelerates from year to year. Of all environmental exposures the one that would most exactly meet such criteria would be a food-borne toxin eaten every day. The excitotoxin food additives would certainly fulfill such a requirement. One would expect neuron loss under such exposure to be rather uniform."


 

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