Date:   3/9/2011 9:21:48 PM ( 13 y ago)
Hits:   7,937
URL:   https://www.curezone.org/forums/fm.asp?i=1781732

Dear friends,

Here's something that many ulcer sufferers may find useful. This is the evidence that the bacteria that causes peptic ulcers (H. pylori) seems to act together with the Candida fungus. So if someone is trying to recover from ulcers, make sure you address the systemic Candida infection in your body as well. There are many ways to do that but, first of all, don't eat any sweets. Because eating Sugar is a sure way to open up your body to Candida.

As the authors point out, a low curability of ulcers infected with Candida has already been known for a while, but now there's scientific proof.

All the best,

eio

Longxue Jin, Masashi Yoshida, et al. Candida albicans Infection Delays Duodenal Ulcer Healing in Cysteamine-Induced Duodenal Ulcers in Rats

Published in: Digestive Diseases and Sciences
Volume 53, Number 11 (2008), 2878-2885,
DOI: 10.1007/s10620-008-0385-9

Abstract
A low curability of ulcers infected with Candida has been reported in the literature. The aim of the study reported here was to investigate experimentally whether Candida infection affects the healing of ulcers. Candida albicans (the Candida group) or saline (the control group) was administered intragastrically into rats with a cysteamine-induced duodenal ulcer. The duodenal lesions, vascular endothelial growth factor A (VEGF-A) and proliferating cell nuclear antigen (PCNA) were assessed. On Day 7 post-administration, 70.4% rats of the Candida group had a duodenal ulcer compared with 33.3% in the control group (P < 0.05). The duodenal ulcer in the Candida group was significantly larger and deeper than that in the control group. The number of VEGF-A- and PCNA-positive cells was smaller and the area of VEGF-A expression was lower in the Candida group. Using a rat model, we have demonstrated that Candida infection can delay the wound healing process of duodenal ulcers by means of a low expression of VEGF-A and PCNA.

 

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