Date:   3/29/2009 2:40:43 AM ( 15 y ago)
Hits:   14,242
URL:   https://www.curezone.org/forums/fm.asp?i=1384957

There's been rumours that taking Miracle-Mineral-Supplement [chlorine dioxide] can deplete thyroid hormones.

I found this old post in another group [can't link to it, sorry].

I'm thinking, if we just take Iodine regularly [which we should anyway], then Miracle-Mineral-Supplement won't have a detrimental impact on the thyroid, as the Iodine will work to restore.

What do you think?


Here's the post:

"Toxicological studies dealing with recent findings of health effects
of drinking water disinfectants are reviewed. Experiments with
monkeys and rodents indicate that the biological activity of ingested
disinfectants is expressed via their chemical interaction with the
mucosal epithelia, secretory products, and nutritional contents of
the alimentary tract. Evidence exists that a principal partner of
this redox interaction is the Iodide of nutritional origin that is
ubiquitous in the gastrointestinal tract. Thus the observation that
subchronic exposure to chlorine dioxide (ClO2) in drinking water
decreases serum thyroxine levels in mammalian species can be best
explained with changes produced in the chemical form of the
bioavailable iodide. Ongoing and previously reported mechanistic
studies indicate that oxidizing agents such as chlorine-based
disinfectants oxidize the basal Iodide content of the
gastrointestinal tract. The resulting reactive Iodine species readily
attaches to organic matter by covalent bonding. Evidence suggests
that the extent to which such iodinated organics are formed is
proportional to the magnitude of the electromotive force and
stoichiometry of the redox couple between Iodide and the
disinfectant. Because the extent of thyroid uptake of the
bioavailable iodide does not decrease during ClO2 ingestion, it seems
that ClO2 does not cause iodide deficiency of sufficient magnitude to
account for the decrease in hormonogenesis. Absorption of one or more
of iodinated molecules, e.g., nutrients, hormones, or cellular
constituents of the alimentary tract having thyromimetic or thyroid
inhibitory properties, is a better hypothesis for the effects seen."
 

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