FANTASTIC FIND!..but it's BROMINE POISONING...NOT excess iodine.

WHY does almost EVERYone continue to blame "excess iodine" for the researched & proven symptoms of high blood levels of bromine???!!! Taking too much Iodine does NOT cause the symptoms below...it's the fact that we've been poisoned by bromine that causes the symptoms! GRRRRRR!

Excess Iodine Symptoms

Acne, rashes
Diarrhea
Headache
Increased salivation
Metallic taste in mouth
Nausea
Sneezing

Below I have compiled a list of gazillions of symptoms we've all experienced...read it and RAGE!! They CONTINUE to put this poison in our food (and it's in the chemtrails they spray on us)! And you can find almost all of the symptoms of 'excess iodine' in the symptoms for bromine poisoning. I don't think I captured the sneezing or metal taste...but I did see them, I just ran out of time to list them all.


http://www.gulflink.osd.mil/library/randrep/pb_paper/mr1018.2.chap10.html

--Intake of excessive bromide can induce a condition termed bromism, with neurological, psychiatric, dermatological, and possibly endocrine effects
--Psychiatric symptoms may include, in the earlier stages, disinhibition, self-neglect, fatigue, sluggishness, impairment of memory and concentration, irritability or emotional instability, and depression. Symptoms of more advanced disease may include confusion but occasionally schizophrenic-like psychotic behavior or hallucinations in clear consciousness. Behavior may become violent, especially at night. There may be severe auditory or visual hallucinations, or both. There may be clouding of consciousness, including stupor and coma.
--Neurological abnormalities of "all kinds" occur, including headache, tremor, slurred speech, spontaneous movements, incoordination, ataxia (abnormal gait), tendon reflex changes (increased or decreased reflexes), and extensor plantar responses (abnormal reflexes that signify "upper motor neuron" disease, disease in the brain or spinal cord that affects nerve signaling to the muscles)
-- Vision changes have included decreased visual acuity often in conjunction with enlarged and poorly reacting pupils; disturbances of color perception, photophobia (abnormal sensitivity to light), micropsia and macropsia (perception of things as smaller or larger than they are), blurring of vision, and the quite characteristic mydriasis (enlarged pupils)
--Dermatologic: Some patients will develop "bromoderma," an acne-like "papular" (raised) eruption of the face and hands (Horowitz, 1997; Wacks, Oster, et al., 1990); a "macular" (nonraised) rash may also be seen
--In one case series, four of six bromism patients had abnormal pigmentation, usually of the sun-exposed areas
--Acute bromism is seldom seen because the bromide ion is irritating to the GI tract and produces vomiting before sufficient blood levels can be reached to cause bromism from short-term use. However, chronic bromism may develop--bromide is excreted slowly through the kidney, giving it the opportunity to build up in the body through ongoing ingestion
--In the presence of clinical suspicion (typically arising from signs or symptoms of neurological or psychiatric disturbance without other identified cause), (1) a careful review of prescription and over-the-counter medications should be undertaken to elicit use of medications that may contain bromide. A dietary history may also be helpful; one report has implicated excessive consumption of colas containing brominated vegetable oil
*-!-* -- Where suspicion directs, (3) serum bromide level should be secured (Wacks, Oster, et al., 1990). A serum bromide concentration above 50 mg/dL is considered confirmatory (Carney, 1973), and significant symptoms usually do not occur below this level (but see section on "Low-Level Bromism," below).
--The most important element of treatment is cessation of bromide exposure. Additional treatment of bromide toxicity has traditionally involved saline loading (administration of large quantities of sodium chloride in water, typically by vein), which enhances kidney excretion of bromide. The chloride ion from sodium chloride competes with and replaces the bromide ion throughout the body.
--Patients with a depressed mental status due to bromism, or with bromide levels in excess of 200 mg/dL (25 mmol/L) may not achieve nontoxic levels (without treatment) for almost a month and would require five days of saline loading to achieve levels below 50 mg/dL (6.3 mmol/L), the threshold of what is normally construed as the toxic level (Horowitz, 1997). Therefore, in these cases it may be appropriate to use hemodialysis to remove the bromide ion from the blood rather than relying on the kidney (Horowitz, 1997).
--Bromide Blood Concentration and Toxicity

mg/dL mEq/L = mmol/L Toxicity
< 50 < 6.3 "Therapeutic"
50-100 6.3-12.5 Possible toxicity
100-200 12.5-25 Usually serious toxicity
200-300 25-37.5 Possible coma
>300 >37.5 Possibly fatal

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http://www.harvestfields.netfirms.com/HerbBooks/02/08/17.htm

-- The salts of bromin most commonly used are potassium, ammonium and sodium bromid. These salts act as depressants and narcotics, particularly to the brain and nervous system. They lessen the sensitiveness of the nerves and their conductivity and are also powerful depressants on the heart and sex organs, often causing loss of sex power.

-- Bromids show in the iris as white or yellowish white discolorations. They appear in the form of a crescent in the upper regions of the iris, indicating that the drug exhibits a special affinity for the brain and sympathetic nervous system. (Color plate, a-e, p.) The more strongly marked this sign in the iris the more symptoms of chronic bromism will be exhibited by the patient. A very noticeable symptom of chronic bromid poisoning is a peculiar Acne form rash.

-- The eruptions on the face and neck may turn into abscesses and ulcers. Frequently the victims of bromism exhibit erythema and copper colored blotches. They also suffer from digestive disturbances. Mental symptoms are very prominent, there being a distinct action on the blood vessels of the brain, these blood vessels contract causing anemia and atrophy of tissues, weakening and loss of memory, defective coordination of muscular activity, difficulty in walking and tremor of the limbs.

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http://veterans.house.gov/hearings/schedule106/nov99/11-16-99/rand.htm


--These effects may include muscle twitching, muscle spasms, weakness or paralysis, and secretions from glands. Consequences may include difficulty in breathing, cramping, feeling of urge to urinate or frequent urination, tearing, runny nose, salivation, increased bronchial secretions, diarrhea, and sweating.

--These effects may include confusion, emotional changes such as depression, sleep alterations, and difficulties with concentration and memory.

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http://www.pdrhealth.com/drug_info/nmdrugprofiles/nutsupdrugs/bro_0044.shtml


--Bromine is a readily volatile, dark-reddish-brown liquid with a strong, disagreeable odor (bromos in Greek means stench) and an irritating effect on the eyes and throat.

--Dietary bromide has also been found to alleviate growth retardation caused by hyperthyroidism

--Potassium bromide had been used as a sedative drug in the United States and is still occasionally used as a sedative and anticonvulsant in Europe. Prolonged intake of potassium bromide can lead to bromide intoxication or bromism.

--Symptoms include nausea, vomiting, slurred speech, memory impairment, drowsiness, irritability, ataxia, tremors, hallucinations, mania, delirium, psychoses, stupor and coma. Skin rashes of various types may occur, and toxic epidermal necrolysis has been reported.


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