This is a lengthy article, DO NOT SURFACE READ. The actual treatment for ebola which will virtually eliminate fatalities, as revealed by a doctor who has worked with ebola, is below.
Here is the treatment, complete with MOA. This is a treatment and not a cure, your immune system wipes out the virus, and the treatment gives your immune system time to do it. Here is what Ebola does that is fatal: It causes the complete removal of all vitamin C from the body. No one actually knows what mechanism is involved in doing this, other than a malfunction that is not permanently destructive to whatever is triggered to remove all vitamin C. All the researchers know is that vitamin C drops to zero and all the symptoms of ebola are consistent with a complete loss of vitamin C.
These viral hemorrhagic fever syndromes share certain clinical features. The Cecil Textbook of Medicine notes that these diseases are characterized by capillary fragility, which translates to easy bleeding, that can frequently lead to severe shock and death. These diseases also tend to consume and/or destroy the platelets, which play an integral role in blood clotting. The clinical presentation of these viral diseases is similar to scurvy, which is also characterized by capillary fragility and a tendency to bleed easily. Characteristic skin lesions develop, which are actually multiple tiny areas of bleeding into the skin that surround the hair follicles. some cases even include bleeding into already healed scars.
In the classic form of scurvy that evolves very slowly from the gradual depletion of vitamin C body stores, the immune system will be sufficiently compromised for infection to claim the patient’s life before the extensive hemorrhage that occurs after all vitamin C stores have been completely exhausted. Ebola virus and the other viral hemorrhagic fevers are much more likely to cause hemorrhaging before any other fatal infection has a chance to become established. This is because the virus so rapidly and totally metabolizes and consumes all available vitamin C in the bodies of the victims that an advanced stage of scurvy is literally produced after only a few days of the disease.
The scurvy is so complete that the blood vessels generally cannot keep from hemorrhaging long enough to allow an infective complication to develop. Also, the viral hemorrhagic fevers typically only take hold and reach epidemic proportions in those populations that would already be expected to have low body stores of vitamin C, such as is found in many of the severely malnourished Africans. In such individuals, an infecting hemorrhagic virus will often wipe out any remaining vitamin C stores before the immune systems can get the upper hand and initiate recovery. When the vitamin C stores are rapidly depleted by large infecting doses of an aggressive virus, the immune system gets similarly depleted and compromised. However, this point is largely academic after hemorrhaging throughout the body has begun.
To date, no viral infection has been demonstrated to be resistant to the proper dosing of vitamin C as classically demonstrated by Klenner. However, not all viruses have been treated with Klenner-sized vitamin C doses, or at least the results have not been published. Ebola viral infection and the other acute viral hemorrhagic fevers appear to be diseases that fall into this category. Because of the seemingly exceptional ability of these viruses to rapidly deplete vitamin C stores, even larger doses of vitamin C would likely be required in order to effectively reverse and eventually cure infections caused by these viruses.
Cathcart (1981), who introduced the concept of bowel tolerance to vitamin C discussed earlier, hypothesized that Ebola and the other acute viral hemorrhagic fevers may well require 500,000 mg of vitamin C daily to reach bowel tolerance! Whether this estimate is accurate, it seems clear as evidenced by the scurvy-like clinical manifestations of these infections that vitamin C dosing must be vigorous and given in extremely high doses. If the disease seems to be winning, then even more vitamin C should be given until symptoms begin to lessen. Obviously, these are viral diseases that would absolutely require high doses of vitamin C intravenously as the initial therapy. The oral administration should begin simultaneously, but the intravenous route should not be abandoned until the clinical response is complete. Death occurs too quickly with the hemorrhagic fevers to be conservative when dosing the vitamin C. (from Vitamin C, Infectious Diseases, and Toxins:Curing the Incurable by Thomas E. Levy MD JD)
All curative agents have a mode of action, or MOA. And if anyone posting medical cures does not know the MOA, they have no idea what they are talking about. Colloidal silver has an MOA that has been known for many decades, yet recently Google has been rigged to bury it with only articles stating "the MOA is being explored and we think it is ___(then disinfo)" and there has to be a reason why this is being done right now, at this point in time with Ebola running amok.
Here is how colloidal silver actually works (its MOA), with first an example: Colloidal silver is to bacteria what cyanide is for all red blooded organisms. In red blooded organisms, cyanide binds with hemoglobin in place of oxygen, and makes it impossible for blood to carry oxygen. With enough cyanide, oxygen starvation via cyanide bonded hemoglobin causes death.
Colloidal silver does the same for bacteria, it binds with the oxygen carriers in bacteria permanently, causing bacteria to quickly die from oxygen starvation. This is the MOA for colloidal silver, which has been clearly known practically forever.
A Text-Book of Materia Medica.
(Characteristic, Analytical, and Comparative.)
By Pr. Allen Corson Cowperthwaite, M. D., PH. D., LL. D.
FERRUM IODATUM. Iodide of Iron.- FeI2. General Analysis.- Combines with the blood state and debility of iron, the cachexia of iodine, rendering it especially useful in scrofulous and chlorotic conditions, and when the system is in an impoverished state. In such persons, its chief curative range is in the female sexual system, where may be present passive congestion, atony, and uterine displacements, with their usual phenomena. Characteristic symptoms. Head.- (Ferrum iodatum) Cephalalgia. Ears.- (Ferrum iodatum) Roaring in the ears. Face.- (Ferrum iodatum) Injected, red ; chlorotic look. (Ars., Ferr.) Abdomen.- (Ferrum iodatum) Fullness even after a little food, as if she had eaten too much (Lyc.) ; a sort of upward pressure ; stuffed feeling as if she could not lean forward. (Nux v.) Urine.- (Ferrum iodatum) Dark-colored, depositing a thick white sediment ; urine scalds. Female Organs.- (Ferrum iodatum) Constant bearing down as if something was coming away ; while sitting, feels as if pushed something up ; she can touch the cervix uteri. (Bell., Lil.) Retroversion of the uterus (clinical). Leucorrhoea like boiled starch ; when the bowels move the discharge is stringy. Itching and soreness of vulva and vagina (Sulph.) ; parts much swollen. Therapeutic Range.- (Ferrum iodatum) Atony and passive congestion of female organs ; uterine displacements ; chlorosis in scrofulous patients ; suppurative stage of tuberculosis ; scrofulous affections in general. Compare.- (Ferrum iodatum) Caul., Ferr., Helon., Sep., Sulph.
http://homeoint.org/seror/cowperthwaite/ferr_i.htm