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Understanding ph for Covid-19 Sp02 issues.
 

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Understanding ph for Covid-19 Sp02 issues.


The Medical profession doesn't do a good job understanding ph. All you'll hear from them is "You can't change the blood's ph". It also doesn't help that they use the logarithmic scale. The tiny difference of such tiny numbers confuses them. Such as 7.35 to 7.45, the "normal" range of blood.

But then, if they understood WHY and HOW the blood "attempts" to stay within the normal range it's very possible that millions of lives could have been spared death from Covid-19.

If we were to give a person sublingual resources (alkalizing minerals) lets say, in the form of a lozenge. The blood would attempt to swing in the alkalizing direction. To prevent this, the blood would carry more oxygen to swing back the ph. So, we have people not getting enough oxygen, what sort of change could we see?

Lets ask ChatGPT this question:

A covid-19 patient has a blood ph of 7.25 with a Sp02 of 85. Buffers are given and raise their ph to 7.45. What will the Sp02 level go to?'

The answer we get:

When the pH of a blood sample increases from 7.25 to 7.45, hemoglobin's affinity for oxygen typically increases due to the leftward shift in the oxygen dissociation curve (the Bohr effect).

At an initial SpO2 of 85% with a pH of 7.25, the patient's hemoglobin is carrying a certain amount of oxygen, but the acidity (low pH) can reduce hemoglobin's ability to bind oxygen efficiently. Raising the pH to 7.45 enhances hemoglobin's affinity for oxygen, which would generally result in an increase in oxygen saturation.

While it's challenging to provide an exact SpO2 level without knowing the corresponding partial pressure of oxygen (PaO2), a general estimate can be made:

At a pH of 7.25 and SpO2 of 85%, the corresponding PaO2 might be around 55-70 mmHg.
When the pH is raised to 7.45, the SpO2 could potentially increase to around 90-95%, assuming the PaO2 remains constant or improves due to better oxygen binding.

Therefore, while the exact increase in SpO2 will depend on individual physiological responses, the significant improvement in pH will likely lead to a notable increase in SpO2, possibly reaching the low to mid-90s percentage range. Continuous monitoring and assessment are essential to determine the precise effect in a clinical setting.

Oh my! From 85% to 90% or even 95%. But wait! How do we know if acidosis might be an issue? After all, it was just a "guess" (snicker) on my part. Lets do a search. Oh look, it's a PubMed Abstract entitled "The Role of Acidosis in the Pathogenesis of Severe Forms of COVID-19" (PMCID: PMC8469745 PMID: 34571729) From it we get "Recently, several studies have shown that acidosis, which is increased acidity in the blood and other body tissues, is often associated with severe COVID-19". Say, perhaps an alkalizing IV should have been the norm?

It's sad that damage to the lungs could very well have been prevented by keeping the ph in check by giving alkalizing minerals.

Say, look at that cool chart "Acidosis Control Strategies":

Increasing the blood buffering capacity
Diet and alkaline drinks (Has any Doctor, ever, given Alkaline Water to a Covid patient?)
Choice of drugs that do not contribute to Acidosis (Good luck with that one!)
Maintenance of normal serum potassium level
Redox balance control (So? You'll never give a Covid-19 patient a glucose IV? Because it would be real stupid if you did? NO sweets for Covid patients)
Early Oxygen therapy

The last is just plain stupid. You "want" to know where the early birds are in terms of Sp02. You manage them with alkaline resources, lozenges, drinks or an IV. Putting them on O2 early allows them to sink deeper into Acidosis. Though it will make the Doctor feel better (like he's doing something) seeing a higher Sp02 level, even though it's....false.

To Your Great Health, Gracefully Savage

Bonus reading: If you run the marathon in 2:01 to 2:10, maybe 2:15, taking alkaline resources during the run will get you there before 2:00. Yes, you'll need to ponder on it.
 

 
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