Gallstones & Liver Flush

If you are considering a liver flush for gallstones
or for gallbladder attacks, you should read this page!

Can some gallstones naturally exit gallbladder?

A gallstone may be able to exit gallbladder, if the smallest diameter of that gallstone is smaller then the smallest diameter of the ducts connecting gallbladder with duodenum: Cystic duct, common bile duct and the sphincter of oddi. (when ducts are maximum stretched).

(See picture above. The cystic duct is narrow throat-like duct that connects gallbladder with the common bile duct.
Often, the smallest diameter of a bile ducts between gallbladder and duodenum is the Cystic duct.

How small is the smallest diameter of the Cystic Duct?

No two people are identical. The smallest diameter of the cystic duct in an adult person can be from 3 to 10 millimeters. Rarely, it can be larger then 1 cm, up to 1.5 cm (15 mm) or about half of an inch. That means that the largest gallstone able to exit gallbladder may not be larger then 3 mm across smallest diameter for some people, or 10 mm across smallest diameter for other people.

Can we dissolve gallstones?

Cholesterol based gallstones can be dissolved with bear bile (Ursodiol, also known as ursodeoxycholic acid and the abbreviation UDCA). Lecithin may help dissolve cholesterol based gallstones. Increasing bile flow may help dissolve gallstones.

It may be impossible to dissolve Calcified gallstones. It may be impossible to dissolve Protein based gallstones.
There are unconfirmed reports that Calcified gallstones were dissolved with Freshly pressed radish juice or with some other vegetable or fruit juice. There are unconfirmed reports that Calcified gallstones were dissolved during a water fast.

Ursodeoxycholic acid goes by the trade names Actigall, Ursosan, Urso, Ursodiol and Urso Forte. In Italy, it is marketed under the name Deursil.
Ursodeoxycholic is naturally obrained from bear bile. Ursodeoxycholic acid can be chemically synthesized and was brought to market by the Montreal-based Axcan Pharma in 1998, which continues to market the drug.
The drug reduces cholesterol absorption and is used to dissolve (cholesterol) gallstones in patients who want an alternative to gallbladder surgery.

The drug is very expensive, however, and if the patient stops taking it, the gallstones tend to recur if the condition that gave rise to their formation does not change. For these reasons, it has not supplanted surgical treatment by cholecystectomy.

It is used to treat primary biliary cirrhosis. In children, its use is not licensed, as its safety and effectiveness is not established.

Ursodeoxycholic acid reduces elevated liver enzyme levels by facilitating bile flow through the liver and protecting liver cells.

The drug is generally not derived from animals.

However, it is believed more than 12,000 bile bears are kept on farms in China, Vietnam and South Korea for the purpose of harvesting ursodeoxycholic acid. Ursodeoxycholic acid is found in large quantities in bear bile.

How do we know that some gall stones can naturally exit gallbladder?

Majority of gallstones that manage to pass through a cystic duct may reach intestines. But, some gallstones will exit the gallbladder and then stuck inside the common bile duct.

Actually, the obstruction of the common bile duct is often caused by gallstones expelled from gallbladder. This in itself is the major proof that gallstones can exit gallbladder.

Reference
"In patients with chronic Pancreatitis, common bile duct obstruction is reported in 3.2-45.6% of patients; however, only 5-10% of all patients with chronic Pancreatitis require operative decompression of the bile duct."

Reference
"Passage of gallstones into the common bile duct occurs in approximately 10-15% of patients with Gallstones. The incidence is thus related to the presence of gallstones, which are very common (10-20% of population)."

Reference
"Jaundice occurs in patients with gall stones when a stone migrates from the gall bladder into the common bile duct..."

Read more here Link to a Source

Can all gallstones naturally exit gallbladder?

No. If a gallstone is larger then the smallest diameter of the ducts connecting gallbladder with duodenum: Cystic duct, common bile duct and the sphincter of oddi, then that gallstone will not be able to exit gallbladder.

Can liver flush help gall stones naturally exit gallbladder?

Liver flush involves drinking a large amount of oil.

After drinking a large amount of oil, liver will produce a large amount of bile, and the bile will be stored inside gallbladder, and will exit gallbladder when the oil start moving from stomach into duodenum.

Large amount of bile moving from gallbladder into duodenum may carry some stones out into duodenum!

That way, liver flush may help get the gallstones out. A single liver flush may not be enough.

Read:
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Are there any risks associated with gallstones and gallbladder flushing?

Risks are small, but 1 out of 150 people with symptomatic gallstones may experience stone stuck inside the cystic duct or stone stuck inside the common bile duct. Source: Reference

1 out of 150 people with symptomatic gallstones who try liver flush may experience serious case of acute pancreatitis and may need emergency medical help! Source: Reference

If you have asymptomatic gallstones, the odds are in the range of 1 out of 500.

Read more here about the risks associated with liver flushing:
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How many people with symptomatic gall stones experience cure after liver flushing?

56 of 121 = 46% Source: Reference

How many people with symptomatic gallstones experience improvement after liver flushing?

55 of 115 = 48% Source: Reference

How many people with symptomatic gallstones regretted liver flushing?

2 of 158 = 1% Source: Reference

How many people with symptomatic gall stones got worse after liver flushing?

4 of 131 = 3% Source: Reference

How many people with symptomatic gallstones got no improvement after liver flushing?

6 of 104 = 6% Source: Reference

How many people with symptomatic gallstones experienced SERIOUS case of acute pancreatitis after liver flushing?

1 of 141 = 1% Source: Reference

But, that person have not selected that he/she regretted liver flushing!

How many people with symptomatic gallstones experienced MILD case of acute pancreatitis after liver flushing?

5 of 147 = 3% Source: Reference

One of them regretted liver flushing!

How many people with symptomatic gallstones experienced STRONG gallbladder attack during a liver flush?

1 of 141 = 1% Source: Reference

But, that person have not selected that he/she regretted liver flushing!

How many people with symptomatic gallstones experienced MEDIUM strong gallbladder attack during a liver flush?

18 of 147 = 12% Source: Reference

One of them regretted liver flushing!

How many people with symptomatic gallstones experienced MILD gallbladder attack during a liver flush?

11 of 147 = 7% Source: Reference

One of them regretted liver flushing!

Other Frequently Asked Questions:

Experience Questions:

Is it safe to drink Epsom Salt (Magnesium Sulfate)?

WARNING!

Make sure you are able to tolerate
Magnesium Sulfate (Epsom Salt)
before you attempt to consume 4 tablespoons.

People unable to tolerate Magnesium Sulfate may suffer negative reaction, even death.

Several people died from the results of Epsom Salt overdose.

Fatal Hypermagnesemia Caused by an Epsom Salt Enema PDF 51.2 KB

Tampabay Nurse error spotlights drug's danger PDF 12.2 KB

Dying To Get Well PDF 2 MB

E Numbers Food Additives Classification PDF 229.6 KB

It is safe to drink Epsom salt. But, one out of 100.000 people may experience poisoning, even death.

About MAGNESIUM SULFATE

Description
Reference Link

Magnesium sulfate reduces striated muscle contractions and blocks peripheral neuromuscular transmission by reducing acetylcholine release at the myoneural junction. In emergency care, magnesium
sulfate is used to manage seizures associated with toxemia of pregnancy. Other uses include uterine relaxation (to inhibit contractions of premature labor), as a bronchodilator after beta-agonist and anticholinergic agents have been used, replacement therapy for magnesium deficiency, as a cathartic to reduce the absorption of poisons from the Gl tract, and in the initial therapy for convulsions. Magnesium sulfate is gaining popularity as an initial treatment in the management of various dysrhythmias, particularly torsades de pointes, and dysrhythmias secondary to a tricyclic antidepressant overdose or digitalis toxicity. The drug is also considered as a class Ila agent (probably helpful) for refractory ventricular fibrillation and ventricular tachycardia after administration of lidocaine or bretylium doses.

Magnesium sulfate is effective for severe acute asthma treated in the emergency department
Reference Link

Intravenous magnesium sulfate reduces the rate of hospital admissions and improves pulmonary function in patients with severe acute asthma treated in the emergency department.
Sources of funding: Canadian Association of Emergency Physicians and National Institutes of Health.

Magnesium sulfate is used to treat pre-eclampsia, eclampsia and preterm labor.
Reference Link

Pre-eclampsia (also known as toxemia and Pregnancy-Induced High Blood Pressure) consists of high blood pressure, protein in the urine and edema (swelling). It can rapidly become severe pre-eclampsia, with very high blood pressure, visual disturbances, failing kidneys and elevated liver enzymes. In rare cases, pre-eclampsia develops into eclampsia, where potentially fatal convulsions occur. It also can become HELLP Syndrome (hemolysis (H), which is the breaking down of red blood cells, elevated liver enzymes (EL), and low platelet count (LP)), which is potentially fatal to both the woman and her baby or babies.

Chenodeoxycholic acid

Chenodeoxycholic acid (also known as chenodesoxycholic acid) is a bile acid. It occurs as a white crystalline substance insoluble in water but soluble in alcohol and acetic acid, with melting point at 165-167 °C. Salts of this carboxylic acid are called chenodeoxycholates. Chenodeoxycholic acid is one of the 4 main organic acids produced by the liver.

Chenodeoxycholic acid is synthesized in the liver from cholesterol. It was first isolated in the domestic goose, hence the 'cheno' portion of its name (Greek: χήνα = goose) [1]

This compound, when altered by bacteria in the colon, will result in conversion to its secondary bile acid known as lithocholic acid. Both of these bile acids, in addition to the others, can be conjugated to taurine or glycine. Conjugation, a function carried out by the liver will result in a lowered pKa and therefore, the compounds will remain ionized. These ionized compounds will stay in the gastrointestinal tract until reaching the ileum where they will be reabsorbed. The purpose of this conjugation is to keep the bile acids in the tract until the end to facilitate lipid digestion all the way to the ileum.

In cases where bacteria overgrow in the small intestine, often due to a blind loop in the intestine retaining chyme in one place, the bacteria will de-conjugate the bile acids and therefore impede fat digestion and absorption. This can lead to steatorrhea.

Chenodeoxycholic acid and cholic acid are the most important human bile acids. Some other mammals synthesize predominantly deoxycholic acid.

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